about
Communication in the heart: the role of the innate immune system in coordinating cellular responses to ischemic injuryOrigin, fate and dynamics of macrophages at central nervous system interfacesExploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.Eptifibatide-induced thrombocytopenia and thrombosis.Induction of lysosomal biogenesis in atherosclerotic macrophages can rescue lipid-induced lysosomal dysfunction and downstream sequelaeThe pancreas anatomy conditions the origin and properties of resident macrophages.Distinct macrophage lineages contribute to disparate patterns of cardiac recovery and remodeling in the neonatal and adult heartSoluble angiotensin-converting enzyme 2 in human heart failure: relation with myocardial function and clinical outcomes.Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo.Increasing serum soluble angiotensin-converting enzyme 2 activity after intensive medical therapy is associated with better prognosis in acute decompensated heart failure.Enterococcal endocarditis presenting as an isolated aortic valve aneurysm: case report and review of literature.Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation.H(2)S--the newest gaseous messenger on the block.Therapeutic targeting of innate immunity in the failing heart.Origin and functions of tissue macrophages.Role of innate and adaptive immune mechanisms in cardiac injury and repair.Chronic Heart Failure and Inflammation: What Do We Really Know?Pseudomonas aeruginosa exoenzyme S induces transcriptional expression of proinflammatory cytokines and chemokines.Microbial products activate monocytic cells through detergent-resistant membrane microdomains.Self-renewing resident arterial macrophages arise from embryonic CX3CR1(+) precursors and circulating monocytes immediately after birth.Primitive Embryonic Macrophages are Required for Coronary Development and Maturation.A CD103+ Conventional Dendritic Cell Surveillance System Prevents Development of Overt Heart Failure during Subclinical Viral Myocarditis.Alternatively Activated Macrophages Drive Browning of White Adipose Tissue in Burns.The Macrophage in Cardiac Homeostasis and Disease: JACC Macrophage in CVD Series (Part 4)Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction.Cardiac Macrophages, Reactive Oxygen Species, and Development of Left Ventricular DysfunctionConventional Dendritic Cells Impair Recovery after Myocardial InfarctionEndocannabinoid signalling: bone marrow monocytes and neutrophils follow their nose into ischaemic tissueMicrobes and genes in heart failureIsolation and Identification of Extravascular Immune Cells of the HeartPublisher Correction: Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarctionTherapeutic Treatment Approaches Post-Myocardial Infarction: A Bias Toward Formyl Peptide Receptor AgonistsNext-Generation Approaches to Predicting the Need for Heart Failure HospitalizationMonocyte-Derived Macrophages: The Missing Link in Organ Transplantation
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Slava Epelman
@ast
Slava Epelman
@en
Slava Epelman
@es
Slava Epelman
@nl
type
label
Slava Epelman
@ast
Slava Epelman
@en
Slava Epelman
@es
Slava Epelman
@nl
prefLabel
Slava Epelman
@ast
Slava Epelman
@en
Slava Epelman
@es
Slava Epelman
@nl
P106
P1153
6601987283
P31
P496
0000-0003-3400-4475