Neuropathology of mice with targeted disruption of Hexa gene, a model of Tay-Sachs disease
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Mouse models of Tay-Sachs and Sandhoff diseases differ in neurologic phenotype and ganglioside metabolismMice lacking both subunits of lysosomal beta-hexosaminidase display gangliosidosis and mucopolysaccharidosisGlycosphingolipid lysosomal storage diseases: therapy and pathogenesis.Sphingolipid metabolism. Sphingoid analogs, sphingolipid activator proteins, and the pathology of the cell.Reduced rates of axonal and dendritic growth in embryonic hippocampal neurones cultured from a mouse model of Sandhoff disease.
P2860
Neuropathology of mice with targeted disruption of Hexa gene, a model of Tay-Sachs disease
description
article
@en
im März 1995 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в березні 1995
@uk
ലേഖനം
@ml
name
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@en
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@nl
type
label
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@en
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@nl
prefLabel
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@en
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@nl
P2093
P356
P1476
Neuropathology of mice with ta ...... , a model of Tay-Sachs disease
@en
P2093
Clarita Langaman
Masako Taniike
Richard L. Proia
Shoji Yamanaka
Teresa Bone-Turrentine
P304
P356
10.1007/S004010050250
P577
1995-03-01T00:00:00Z