Hydrogen peroxide-induced MAPK activation causes the increase of RCAN1 (DSCR1) protein expression

about

Hydrogen peroxide-induced MAPK activation causes the increase of RCAN1 (DSCR1) protein expression

Item

http://www.wikidata.org/entity/Q56602773

description

wetenschappelijk artikel

@nl

наукова стаття, опублікована в січні 2013

@uk

name

Hydrogen peroxide-induced MAPK ...... AN1 (DSCR1) protein expression

@en

Hydrogen peroxide-induced MAPK activation causes the increase of RCAN1

@nl

type

Item

label

Hydrogen peroxide-induced MAPK ...... AN1 (DSCR1) protein expression

@en

Hydrogen peroxide-induced MAPK activation causes the increase of RCAN1

@nl

prefLabel

Hydrogen peroxide-induced MAPK ...... AN1 (DSCR1) protein expression

@en

Hydrogen peroxide-induced MAPK activation causes the increase of RCAN1

@nl

P1476

Hydrogen peroxide-induced MAPK ...... AN1 (DSCR1) protein expression

@en

P2093

Seon Sook Kim

http://www.w3.org/2001/XMLSchema#string

Su Ryeon Seo

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphorylation of calcipressin 1 increases its ability to inhibit calcineurin and decreases calcipressin half-life

The SCFCdc4 ubiquitin ligase regulates calcineurin signaling through degradation of phosphorylated Rcn1, an inhibitor of calcineurin.

DSCR1, overexpressed in Down syndrome, is an inhibitor of calcineurin-mediated signaling pathways

Chronic overexpression of the calcineurin inhibitory gene DSCR1 (Adapt78) is associated with Alzheimer's disease

Multiple roles of the DSCR1 (Adapt78 or RCAN1) gene and its protein product calcipressin 1 (or RCAN1) in disease

NF-kappaB-inducing kinase phosphorylates and blocks the degradation of Down syndrome candidate region 1.

Alzheimer's disease and down syndrome: from meiosis to dementia.

Regulator of calcineurin 1 (RCAN1) facilitates neuronal apoptosis through caspase-3 activation.

The Drosophila homolog of Down's syndrome critical region 1 gene regulates learning: implications for mental retardation.

Oxidative stress, cell cycle, and neurodegeneration.

P2888

s13258-013-0080-x

P304

111-116

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S13258-013-0080-X

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2013-01-29T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

257800669

http://www.w3.org/2001/XMLSchema#string