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SAP97 and dystrophin macromolecular complexes determine two pools of cardiac sodium channels Nav1.5 in cardiomyocytesDifferent isoforms of synapse-associated protein, SAP97, are expressed in the heart and have distinct effects on the voltage-gated K+ channel Kv1.5Multidrug resistance-associated protein 4 regulates cAMP-dependent signaling pathways and controls human and rat SMC proliferationCardiac-specific ablation of synapse-associated protein SAP97 in mice decreases potassium currents but not sodium currentDoxorubicin induces slow ceramide accumulation and late apoptosis in cultured adult rat ventricular myocytesLong-term outcomes of pandemic 2009 influenza A(H1N1)-associated severe ARDS.Shear stress triggers insertion of voltage-gated potassium channels from intracellular compartments in atrial myocytes.Dominant-negative effect of SCN5A N-terminal mutations through the interaction of Na(v)1.5 α-subunitsModerate and chronic hemodynamic overload of sheep atria induces reversible cellular electrophysiologic abnormalities and atrial vulnerability.A truncating SCN5A mutation combined with genetic variability causes sick sinus syndrome and early atrial fibrillation.Peroxisome proliferator-activated receptor beta stimulation induces rapid cardiac growth and angiogenesis via direct activation of calcineurin.The antiarrhythmic agent bertosamil induces inactivation of the sustained outward K+ current in human atrial myocytes.Regulation of cAMP homeostasis by the efflux protein MRP4 in cardiac myocytes.Mutation of delta-sarcoglycan is associated with Ca(2+) -dependent vascular remodeling in the Syrian hamster.Mesenchymal stem cell delivery into rat infarcted myocardium using a porous polysaccharide-based scaffold: a quantitative comparison with endocardial injection.Cholesterol modulates the recruitment of Kv1.5 channels from Rab11-associated recycling endosome in native atrial myocytes.Specificities of atrial electrophysiology: Clues to a better understanding of cardiac function and the mechanisms of arrhythmias.Cholesterol and cardiac arrhythmias.Dynamic of ion channel expression at the plasma membrane of cardiomyocytes.Epicardial adipose tissue and atrial fibrillation.The European Network for Translational Research in Atrial Fibrillation (EUTRAF): objectives and initial results.A roadmap to improve the quality of atrial fibrillation management: proceedings from the fifth Atrial Fibrillation Network/European Heart Rhythm Association consensus conference.Novel mechanisms in the pathogenesis of atrial fibrillation: practical applications.Expert consensus document: Defining the major health modifiers causing atrial fibrillation: a roadmap to underpin personalized prevention and treatment.Cardiac adipose tissue and atrial fibrillation: the perils of adiposity.Somatotopic organization of the analgesic effects of motor cortex rTMS in neuropathic pain.Left atrial aging: a cardiac magnetic resonance feature-tracking study.Human epicardial adipose tissue has a specific transcriptomic signature depending on its anatomical peri-atrial, peri-ventricular, or peri-coronary location.Assessment of left atrial function by MRI myocardial feature tracking.SERCA2a controls the mode of agonist-induced intracellular Ca2+ signal, transcription factor NFAT and proliferation in human vascular smooth muscle cells.Membrane cholesterol modulates Kv1.5 potassium channel distribution and function in rat cardiomyocytes.The anchoring protein SAP97 retains Kv1.5 channels in the plasma membrane of cardiac myocytes.Atrial fibrillation and rapid acute pacing regulate adipocyte/adipositas-related gene expression in the atria.Dedifferentiation of atrial myocytes during atrial fibrillation: role of fibroblast proliferation in vitro.Expression, regulation and role of the MAGUK protein SAP-97 in human atrial myocardium.Overexpression of cAMP-response element modulator causes abnormal growth and development of the atrial myocardium resulting in a substrate for sustained atrial fibrillation in mice.Maximal exercise limitation in functionally overreached triathletes: role of cardiac adrenergic stimulation.Statin, the black box.Downregulation of the calcium current in human right atrial myocytes from patients in sinus rhythm but with a high risk of atrial fibrillation.Spironolactone reduces fibrosis of dilated atria during heart failure in rats with myocardial infarction.
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Stéphane Hatem
@ast
Stéphane Hatem
@en
Stéphane Hatem
@es
Stéphane Hatem
@nl
type
label
Stéphane Hatem
@ast
Stéphane Hatem
@en
Stéphane Hatem
@es
Stéphane Hatem
@nl
prefLabel
Stéphane Hatem
@ast
Stéphane Hatem
@en
Stéphane Hatem
@es
Stéphane Hatem
@nl
P106
P1153
7005197118
P21
P31
P496
0000-0002-9790-0755