The role of CTLA-4 in tolerance induction and ttigen administration cell differentiation in experimental autoimmune encephalomyelitis: i. v. antigen administration
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Mechanisms of immunological tolerance in central nervous system inflammatory demyelination.MOG(35-55) i.v suppresses experimental autoimmune encephalomyelitis partially through modulation of Th17 and JAK/STAT pathways.Immune tolerance induced by intravenous transfer of immature dendritic cells via up-regulating numbers of suppressive IL-10(+) IFN-γ(+)-producing CD4(+) T cells.
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The role of CTLA-4 in tolerance induction and ttigen administration cell differentiation in experimental autoimmune encephalomyelitis: i. v. antigen administration
description
im Dezember 1999 veröffentlichter wissenschaftlicher Artikel
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scientific article published on 01 December 1999
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wetenschappelijk artikel
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наукова стаття, опублікована в грудні 1999
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name
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
@en
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
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type
label
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
@en
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
@nl
prefLabel
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
@en
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
@nl
P2093
P356
P1476
The role of CTLA-4 in toleranc ...... : i. v. antigen administration
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P2093
A E Lovett-Racke
L R Arredondo
P J Perrin
P304
P356
10.1093/INTIMM/11.12.1889
P407
P577
1999-12-01T00:00:00Z