about
The Mitochondrial Phosphatase PGAM5 Is Dispensable for Necroptosis but Promotes Inflammasome Activation in Macrophages.RIPK1 and PGAM5 Control Leishmania Replication through Distinct Mechanisms.Cell-Extrinsic TNF Collaborates with TRIF Signaling To Promote Yersinia-Induced Apoptosis.RIPK1-dependent apoptosis bypasses pathogen blockade of innate signaling to promote immune defense.Kinase Activities of RIPK1 and RIPK3 Can Direct IFN-β Synthesis Induced by Lipopolysaccharide.Pathogen blockade of TAK1 triggers caspase-8-dependent cleavage of gasdermin D and cell deathLUBAC prevents lethal dermatitis by inhibiting cell death induced by TNF, TRAIL and CD95L
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Peter J Gough
@ast
Peter J Gough
@en
Peter J Gough
@es
Peter J Gough
@nl
type
label
Peter J Gough
@ast
Peter J Gough
@en
Peter J Gough
@es
Peter J Gough
@nl
prefLabel
Peter J Gough
@ast
Peter J Gough
@en
Peter J Gough
@es
Peter J Gough
@nl
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P31
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0000-0002-5110-9300