about
Troponin T3 expression in skeletal and smooth muscle is required for growth and postnatal survival: characterization of Tnnt3(tm2a(KOMP)Wtsi) miceAn FGF-WNT gene regulatory network controls lung mesenchyme developmentSequential roles of Hedgehog and Wnt signaling in osteoblast developmentAn integrated physical map of 8q22-q24: use in positional cloning and deletion analysis of Langer-Giedion syndrome.Efficacy of colistin-impregnated beads to prevent multidrug-resistant A. baumannii implant-associated osteomyelitis.RBP-Jκ-dependent Notch signaling is required for murine articular cartilage and joint maintenance.Delayed fracture healing and increased callus adiposity in a C57BL/6J murine model of obesity-associated type 2 diabetes mellitus.RBPjkappa-dependent Notch signaling regulates mesenchymal progenitor cell proliferation and differentiation during skeletal developmentNOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB.Chronic axial compression of the mouse tail segment induces MRI bone marrow edema changes that correlate with increased marrow vasculature and cellularity.Daily oral consumption of hydrolyzed type 1 collagen is chondroprotective and anti-inflammatory in murine posttraumatic osteoarthritis.NOTCH-Mediated Maintenance and Expansion of Human Bone Marrow Stromal/Stem Cells: A Technology Designed for Orthopedic Regenerative Medicine.TNF is required for the induction but not the maintenance of compression-induced BME signals in murine tail vertebrae: limitations of anti-TNF therapy for degenerative disc diseaseTransient gamma-secretase inhibition accelerates and enhances fracture repair likely via Notch signaling modulationTAK1 regulates cartilage and joint development via the MAPK and BMP signaling pathways.BMP2, but not BMP4, is crucial for chondrocyte proliferation and maturation during endochondral bone development.Teriparatide as a chondroregenerative therapy for injury-induced osteoarthritis.Cartilage-specific RBPjκ-dependent and -independent Notch signals regulate cartilage and bone developmentA dual role for NOTCH signaling in joint cartilage maintenance and osteoarthritisNotch signaling controls chondrocyte hypertrophy via indirect regulation of Sox9.PTH receptor signaling in osteoblasts regulates endochondral vascularization in maintenance of postnatal growth plate.Rac1 activation controls nuclear localization of beta-catenin during canonical Wnt signaling.Notch signaling in postnatal joint chondrocytes, but not subchondral osteoblasts, is required for articular cartilage and joint maintenanceNOTCH signaling in skeletal progenitors is critical for fracture repair.CCN1 Regulates Chondrocyte Maturation and Cartilage Development.Notch signaling indirectly promotes chondrocyte hypertrophy via regulation of BMP signaling and cell cycle arrest.Suppressive Effects of Insulin on Tumor Necrosis Factor-Dependent Early Osteoarthritic Changes Associated With Obesity and Type 2 Diabetes Mellitus.HES factors regulate specific aspects of chondrogenesis and chondrocyte hypertrophy during cartilage developmentRegulation of chondrogenesis and chondrocyte differentiation by stress.Tamoxifen-inducible gene deletion reveals a distinct cell type associated with trabecular bone, and direct regulation of PTHrP expression and chondrocyte morphology by Ihh in growth region cartilage.Suppression of CXCL12 production by bone marrow osteoblasts is a common and critical pathway for cytokine-induced mobilizationNotch signaling maintains bone marrow mesenchymal progenitors by suppressing osteoblast differentiation.TAK1 regulates SOX9 expression in chondrocytes and is essential for postnatal development of the growth plate and articular cartilages.Mechanism of shortened bones in mucopolysaccharidosis VIIImpact of Smad3 loss of function on scarring and adhesion formation during tendon healing.Multiple hereditary exostoses (MHE): elucidating the pathogenesis of a rare skeletal disorder through interdisciplinary research.Engineering superficial zone features in tissue engineered cartilage.Use of Hes1-GFP reporter mice to assess activity of the Hes1 promoter in bone cells under chronic inflammation.Ski inhibits TGF-β/phospho-Smad3 signaling and accelerates hypertrophic differentiation in chondrocytesThe effect of mesenchymal stem cell sheets on structural allograft healing of critical sized femoral defects in mice.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Matthew J Hilton
@ast
Matthew J Hilton
@en
Matthew J Hilton
@es
Matthew J Hilton
@nl
type
label
Matthew J Hilton
@ast
Matthew J Hilton
@en
Matthew J Hilton
@es
Matthew J Hilton
@nl
prefLabel
Matthew J Hilton
@ast
Matthew J Hilton
@en
Matthew J Hilton
@es
Matthew J Hilton
@nl
P106
P1153
9041749400
P31
P496
0000-0003-3165-267X