about
Aspirin induces apoptosis through the inhibition of proteasome functionCo-chaperone CHIP stabilizes aggregate-prone malin, a ubiquitin ligase mutated in Lafora disease.Grb2 is regulated by foxd3 and has roles in preventing accumulation and aggregation of mutant huntingtinRecent advances in understanding the pathogenesis of polyglutamine diseases: involvement of molecular chaperones and ubiquitin-proteasome pathway.Malin regulates Wnt signaling pathway through degradation of dishevelled2Understanding the pathogenesis of Angelman syndrome through animal modelsDysfunction of the ubiquitin ligase Ube3a may be associated with synaptic pathophysiology in a mouse model of Huntington diseaseDysregulation of core components of SCF complex in poly-glutamine disordersNeuronatin-mediated aberrant calcium signaling and endoplasmic reticulum stress underlie neuropathology in Lafora diseaseRole of ubiquitin protein ligases in the pathogenesis of polyglutamine diseases.Azadiradione ameliorates polyglutamine expansion disease in Drosophila by potentiating DNA binding activity of heat shock factor 1.MicroRNA-124 targets CCNA2 and regulates cell cycle in STHdh(Q111)/Hdh(Q111) cells.Dexamethasone induces heat shock response and slows down disease progression in mouse and fly models of Huntington's disease.UBE3A/E6-AP regulates cell proliferation by promoting proteasomal degradation of p27.Regulation of turnover of tumor suppressor p53 and cell growth by E6-AP, a ubiquitin protein ligase mutated in Angelman mental retardation syndrome.The co-chaperone CHIP is induced in various stresses and confers protection to cells.Oxidative stress promotes mutant huntingtin aggregation and mutant huntingtin-dependent cell death by mimicking proteasomal malfunction.Curcumin induces stress response, neurite outgrowth and prevent NF-kappaB activation by inhibiting the proteasome function.BAG-1 associates with the polyglutamine-expanded huntingtin aggregates.Co-chaperone CHIP associates with expanded polyglutamine protein and promotes their degradation by proteasomes.Inhibition of proteasomal function by curcumin induces apoptosis through mitochondrial pathway.Pro-apoptotic protein kinase C delta is associated with intranuclear inclusions in a transgenic model of Huntington's disease.Comparative effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on MCF-7, RL95-2, and LNCaP cells: role of target steroid hormones in cellular responsiveness to CYP1A1 induction.E6-AP association promotes SOD1 aggresomes degradation and suppresses toxicity.Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates.Deficiency of Ube3a in Huntington's disease mice brain increases aggregate load and accelerates disease pathology.Trehalose-Functionalized Gold Nanoparticle for Inhibiting Intracellular Protein Aggregation.Strain differences in cytochrome P4501A1 gene expression caused by 2,3,7,8-tetrachlorodibenzo-p-dioxin in the rat liver: role of the aryl hydrocarbon receptor and its nuclear translocator.Reversal of reduced parvalbumin neurons in hippocampus and amygdala of Angelman syndrome model mice by chronic treatment of fluoxetine.Abundant secretory lipocalins displaying male and lactation-specific expression in adult hamster submandibular gland. cDNA cloning and sex hormone-regulated repression.Sugar-Terminated Nanoparticle Chaperones Are 102-105 Times Better Than Molecular Sugars in Inhibiting Protein Aggregation and Reducing Amyloidogenic Cytotoxicity.The E3 ligase ube3a is required for learning in Drosophila melanogaster.Poly(trehalose) Nanoparticles Prevent Amyloid Aggregation and Suppress Polyglutamine Aggregation in a Huntington's Disease Model Mouse.The ubiquitin ligase E6-AP promotes degradation of alpha-synuclein.Environmental Enrichment Improves Behavioral Abnormalities in a Mouse Model of Angelman Syndrome.Defective glucocorticoid hormone receptor signaling leads to increased stress and anxiety in a mouse model of Angelman syndrome.Induction of chemokines, MCP-1, and KC in the mutant huntingtin expressing neuronal cells because of proteasomal dysfunction.Impaired adult hippocampal neurogenesis and its partial reversal by chronic treatment of fluoxetine in a mouse model of Angelman syndrome.Role of Estradiol Receptor-α in Differential Expression of 2,3,7,8-Tetrachlorodibenzo-p-dioxin-Inducible Genes in the RL95-2 and KLE Human Endometrial Cancer Cell LinesThyroid hormone induces a 52 kDa soluble protein in goat testis Leydig cell which stimulates androgen release
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wetenschapper
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հետազոտող
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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Nihar Jana
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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Nihar R Jana
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P106
P1153
7006695923
P31
P496
0000-0002-6549-4211