about
Smad6 and Smad7 are co-regulated with hepcidin in mouse models of iron overloadTumor hypoxia modulates podoplanin/CCL21 interactions in CCR7+ NK cell recruitment and CCR7+ tumor cell mobilization.Unbiased RNAi screen for hepcidin regulators links hepcidin suppression to proliferative Ras/RAF and nutrient-dependent mTOR signaling.Imatinib and spironolactone suppress hepcidin expression.SMAD7 controls iron metabolism as a potent inhibitor of hepcidin expression.Bone morphogenetic protein (BMP)-responsive elements located in the proximal and distal hepcidin promoter are critical for its response to HJV/BMP/SMAD.Hepcidin is regulated by promoter-associated histone acetylation and HDAC3.15d-PGJ2 upregulates synthesis of IL-8 in endothelial cells through induction of oxidative stress.Hypoxia induced downregulation of hepcidin is mediated by platelet derived growth factor BB.Cellular citrate levels establish a regulatory link between energy metabolism and the hepatic iron hormone hepcidin
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description
researcher
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wetenschapper
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հետազոտող
@hy
name
Katarzyna Mleczko-Sanecka
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Katarzyna Mleczko-Sanecka
@en
Katarzyna Mleczko-Sanecka
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Katarzyna Mleczko-Sanecka
@nl
type
label
Katarzyna Mleczko-Sanecka
@ast
Katarzyna Mleczko-Sanecka
@en
Katarzyna Mleczko-Sanecka
@es
Katarzyna Mleczko-Sanecka
@nl
prefLabel
Katarzyna Mleczko-Sanecka
@ast
Katarzyna Mleczko-Sanecka
@en
Katarzyna Mleczko-Sanecka
@es
Katarzyna Mleczko-Sanecka
@nl
P214
P106
P214
P31
P496
0000-0001-9095-9597
P7859
viaf-254901913