about
Disruption of the Gut Microbiome: Clostridium difficile Infection and the Threat of Antibiotic ResistanceThe FxRxHrS motif: a conserved region essential for DNA binding of the VirR response regulator from Clostridium perfringens.Functional analysis of the VirSR phosphorelay from Clostridium perfringens.Utility of the clostridial site-specific recombinase TnpX to clone toxic-product-encoding genes and selectively remove genomic DNA fragments.The cysteine protease α-clostripain is not essential for the pathogenesis of Clostridium perfringens-mediated myonecrosis.Towards an understanding of the role of Clostridium perfringens toxins in human and animal disease.Perfringolysin O expression in Clostridium perfringens is independent of the upstream pfoR gene.TcsL is an essential virulence factor in Clostridium sordellii ATCC 9714Skewed genomic variability in strains of the toxigenic bacterial pathogen, Clostridium perfringens.The pore-forming α-toxin from clostridium septicum activates the MAPK pathway in a Ras-c-Raf-dependent and independent mannerClostridium sordellii genome analysis reveals plasmid localized toxin genes encoded within pathogenicity lociThe NEAT Domain-Containing Proteins of Clostridium perfringens Bind Heme.The Sialidase NanS Enhances Non-TcsL Mediated Cytotoxicity of Clostridium sordelliiThe NanI and NanJ sialidases of Clostridium perfringens are not essential for virulence.Antibiotic resistance, virulence factors and genetics of Clostridium sordellii.Functional analysis of an feoB mutant in Clostridium perfringens strain 13.Necrotic Enteritis in Chickens Associated with Clostridium sordellii.Alpha-toxin of Clostridium perfringens is not an essential virulence factor in necrotic enteritis in chickens.Structural Characterization of Clostridium sordellii Spores of Diverse Human, Animal, and Environmental Origin and Comparison to Clostridium difficile Spores.Clostridium perfringens alpha toxin affects electrophysiological properties of isolated jejunal mucosa of laying hens.TcdB or not TcdB: a tale of two Clostridium difficile toxins.Opioid analgesics stop the development of clostridial gas gangrene.Expression of the large clostridial toxins is controlled by conserved regulatory mechanisms.Clostridium sordellii Pathogenicity Locus Plasmid pCS1-1 Encodes a Novel Clostridial Conjugation Locus.Clostridium sordellii outer spore proteins maintain spore structural integrity and promote bacterial clearance from the gastrointestinal tract.The α-toxin ofClostridium septicumis essential for virulencePaeniclostridium sordellii and Clostridioides difficile encode similar and clinically relevant tetracycline resistance loci in diverse genomic locationsLectin Activity of the TcdA and TcdB Toxins of Clostridium difficilepCP13, a representative of a new family of conjugative toxin plasmids in Clostridium perfringensTranexamic Acid Influences the Immune Response, but not Bacterial Clearance in a Model of Post-Traumatic Brain Injury PneumoniaCephamycins inhibit pathogen sporulation and effectively treat recurrent Clostridioides difficile infectionComparing the identification of Clostridium spp. by two Matrix-Assisted Laser Desorption Ionization-Time of Flight (MALDI-TOF) mass spectrometry platforms to 16S rRNA PCR sequencing as a reference standard: a detailed analysis of age of culture and
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Milena Awad
@ast
Milena Awad
@en
Milena Awad
@es
Milena Awad
@nl
type
label
Milena Awad
@ast
Milena Awad
@en
Milena Awad
@es
Milena Awad
@nl
prefLabel
Milena Awad
@ast
Milena Awad
@en
Milena Awad
@es
Milena Awad
@nl
P106
P1153
7102519081
P31
P496
0000-0002-9275-3979