about
Candida albicans interactions with epithelial cells and mucosal immunityIdentification of citrullinated alpha-enolase as a candidate autoantigen in rheumatoid arthritisEvaluation of the role of Candida albicans agglutinin-like sequence (Als) proteins in human oral epithelial cell interactionsOral epithelial cells and their interactions with HIV-1.Oral and vaginal epithelial cell lines bind and transfer cell-free infectious HIV-1 to permissive cells but are not productively infected.Candida albicans yeast and hyphae are discriminated by MAPK signaling in vaginal epithelial cells.A biphasic innate immune MAPK response discriminates between the yeast and hyphal forms of Candida albicans in epithelial cells.Candidalysin is a fungal peptide toxin critical for mucosal infectionCandida albicans cell wall glycosylation may be indirectly required for activation of epithelial cell proinflammatory responses.Adaptive immune responses to Candida albicans infection.Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface.Insertional polymorphisms: a new lease of life for endogenous retroviruses in human disease.Clotrimazole dampens vaginal inflammation and neutrophil infiltration in response to Candida albicans infection.Epithelial discrimination of commensal and pathogenic Candida albicans.Innate immunity in HIV-1 infection: epithelial and non-specific host factors of mucosal immunity- a workshop report.The gut and oral microbiome in HIV disease: a workshop report.The distribution of the endogenous retroviruses HERV-K113 and HERV-K115 in health and disease.The Role of ErbB Receptors in Infection.Analysis of host-cell responses by immunoblotting, ELISA, and real-time PCR.Candidalysin drives epithelial signaling, neutrophil recruitment, and immunopathology at the vaginal mucosa.Processing of Candida albicans Ece1p Is Critical for Candidalysin Maturation and Fungal Virulence.Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin.The Human Mucosal Mycobiome and Fungal Community Interactions.A pilot study of the gingival response when smokers switch from smoking to vaping.IL-36 and IL-1/IL-17 Drive Immunity to Oral Candidiasis via Parallel MechanismsHERV-K113 Is Not Associated with Multiple Sclerosis in a Large Family-Based Study
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
David L Moyes
@ast
David L Moyes
@en
David L Moyes
@es
David L Moyes
@nl
type
label
David L Moyes
@ast
David L Moyes
@en
David L Moyes
@es
David L Moyes
@nl
prefLabel
David L Moyes
@ast
David L Moyes
@en
David L Moyes
@es
David L Moyes
@nl
P106
P21
P31
P496
0000-0002-1657-918X