about
Possible involvement of self-defense mechanisms in the preferential vulnerability of the striatum in Huntington's diseaseHigh throughput screening for inhibitors of REST in neural derivatives of human embryonic stem cells reveals a chemical compound that promotes expression of neuronal genes.Loss of the thyroid hormone-binding protein Crym renders striatal neurons more vulnerable to mutant huntingtin in Huntington's disease.The mechanism of sirtuin 2-mediated exacerbation of alpha-synuclein toxicity in models of Parkinson disease.Contribution of Neuroepigenetics to Huntington's Disease.Identification of a conserved gene signature associated with an exacerbated inflammatory environment in the hippocampus of aging rats.Biological features of herpes simplex virus type 1 latency in mice according to experimental conditions and type of neurones.Correction: The mechanism of sirtuin 2-mediated exacerbation of alpha-synuclein toxicity in models of Parkinson disease.In vivo imaging of brain glutamate defects in a knock-in mouse model of Huntington's disease.Mutant huntingtin alters Tau phosphorylation and subcellular distribution.Striatal long noncoding RNA Abhd11os is neuroprotective against an N-terminal fragment of mutant huntingtin in vivo.Genetic and functional abnormalities of the melatonin biosynthesis pathway in patients with bipolar disorder.The striatal kinase DCLK3 produces neuroprotection against mutant huntingtin.
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Laetitia Francelle
@ast
Laetitia Francelle
@en
Laetitia Francelle
@es
Laetitia Francelle
@nl
type
label
Laetitia Francelle
@ast
Laetitia Francelle
@en
Laetitia Francelle
@es
Laetitia Francelle
@nl
prefLabel
Laetitia Francelle
@ast
Laetitia Francelle
@en
Laetitia Francelle
@es
Laetitia Francelle
@nl
P106
P31
P496
0000-0003-0831-4007