about
Origins of increased airway smooth muscle mass in asthma.IL-33 drives airway hyper-responsiveness through IL-13-mediated mast cell: airway smooth muscle crosstalk.Nociceptin/orphanin FQ (N/OFQ) modulates immunopathology and airway hyperresponsiveness representing a novel target for the treatment of asthmaRationale and clinical results of inhibiting interleukin-5 for the treatment of severe asthma.Asthma therapy and its effect on airway remodelling.Associations in asthma between quantitative computed tomography and bronchial biopsy-derived airway remodelling.Myeloid-derived suppressor cell-like fibrocytes are increased and associated with preserved lung function in chronic obstructive pulmonary disease.Airway smooth muscle NOX4 is upregulated and modulates ROS generation in COPD.Fevipiprant, a prostaglandin D2 receptor 2 antagonist, in patients with persistent eosinophilic asthma: a single-centre, randomised, double-blind, parallel-group, placebo-controlled trial.In vitro, in silico and in vivo study challenges the impact of bronchial thermoplasty on acute airway smooth muscle mass loss.In vivo imaging reveals increased eosinophil uptake in the lungs of obese asthmatic patients
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description
researcher ORCID: 0000-0002-0513-0627
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wetenschapper
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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R Berair
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55754876300
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0000-0002-0513-0627