about
Use of fluroquinolone and risk of Achilles tendon rupture: a population-based cohort studyAnti-TNF treatment response in rheumatoid arthritis patients is associated with genetic variation in the NLRP3-inflammasomePolymorphisms in the inflammatory pathway genes TLR2, TLR4, TLR9, LY96, NFKBIA, NFKB1, TNFA, TNFRSF1A, IL6R, IL10, IL23R, PTPN22, and PPARG are associated with susceptibility of inflammatory bowel disease in a Danish cohort.Polymorphisms in the Toll-Like Receptor and the IL-23/IL-17 Pathways Were Associated with Susceptibility to Inflammatory Bowel Disease in a Danish Cohort.Genetic Variations in Pattern Recognition Receptor Loci Are Associated with Anti-TNF Response in Patients with Rheumatoid Arthritis.Systematic review and meta-analysis: pharmacogenetics of anti-TNF treatment response in rheumatoid arthritis.Genetically determined high activity of IL-12 and IL-18 in ulcerative colitis and TLR5 in Crohns disease were associated with non-response to anti-TNF therapy.Confirmation of an IRAK3 polymorphism as a genetic marker predicting response to anti-TNF treatment in rheumatoid arthritis.Associations between functional polymorphisms in the NFκB signaling pathway and response to anti-TNF treatment in Danish patients with inflammatory bowel disease.Mitochondrial haplogroups in patients with rheumatoid arthritis: No association with disease and disease manifestations.Plasma MicroRNA Profiles in Patients with Early Rheumatoid Arthritis Responding to Adalimumab plus Methotrexate vs Methotrexate Alone: A Placebo-controlled Clinical Trial.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Jacob Sode
@ast
Jacob Sode
@en
Jacob Sode
@es
Jacob Sode
@nl
type
label
Jacob Sode
@ast
Jacob Sode
@en
Jacob Sode
@es
Jacob Sode
@nl
prefLabel
Jacob Sode
@ast
Jacob Sode
@en
Jacob Sode
@es
Jacob Sode
@nl
P106
P31
P496
0000-0002-2718-9320