RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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наукова стаття, опублікована у 2018

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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RtxA Is a Major Factor Driving Inflammatory T Helper Type 17 Cell Responses and

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P1476

Vibrio vulnificus RtxA Is a Ma ...... Responses in vitro and in vivo

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P2093

Arim Lee

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Daeho Cho

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Myun Soo Kim

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Sang Ho Choi

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Tae Sung Kim

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P2860

RTX proteins: a highly diverse family secreted by a common mechanism

The pUC plasmids, an M13mp7-derived system for insertion mutagenesis and sequencing with synthetic universal primers

Generation of large numbers of dendritic cells from mouse bone marrow cultures supplemented with granulocyte/macrophage colony-stimulating factor

Adenylate cyclase toxin from Bordetella pertussis synergizes with lipopolysaccharide to promote innate interleukin-10 production and enhances the induction of Th2 and regulatory T cells.

Identification and characterization of the Vibrio vulnificus rtxA essential for cytotoxicity in vitro and virulence in mice.

Vibrio vulnificus rtxE is important for virulence, and its expression is induced by exposure to host cells.

Construction and phenotypic evaluation of a Vibrio vulnificus vvpE mutant for elastolytic protease.

Regulation of cytotoxicity by quorum-sensing signaling in Vibrio vulnificus is mediated by SmcR, a repressor of hlyU.

Isolation and characterization of a Vibrio vulnificus mutant deficient in both extracellular metalloprotease and cytolysin

Additive function of Vibrio vulnificus MARTX(Vv) and VvhA cytolysins promotes rapid growth and epithelial tissue necrosis during intestinal infection.

P304

2095

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P31

scholarly article

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10.3389/FIMMU.2018.02095

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P407

English

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P50

Kyung Ku Jang

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2018-09-19T00:00:00Z

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P698

30283443

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P932

6157323

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