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The arthritis severity locus Cia5a regulates the expression of inflammatory mediators including Syk pathway genes and proteases in pristane-induced arthritis.Microarray analyses of peripheral blood cells identifies unique gene expression signature in psoriatic arthritis.Increased synovial expression of nuclear receptors correlates with protection in pristane-induced arthritis: a possible novel genetically regulated homeostatic mechanism.Thermal signature analysis as a novel method for evaluating inflammatory arthritis activity.Identification of two new arthritis severity loci that regulate levels of autoantibodies, interleukin-1β, and joint damage in pristane- and collagen-induced arthritisGenetic regulation of T regulatory, CD4, and CD8 cell numbers by the arthritis severity loci Cia5a, Cia5d, and the MHC/Cia1 in the ratSynovial expression of Th17-related and cancer-associated genes is regulated by the arthritis severity locus Cia10.Cia25 on rat chromosome 12 regulates severity of autoimmune arthritis induced with pristane and with collagenCia5d regulates a new fibroblast-like synoviocyte invasion-associated gene expression signature.A resource for the simultaneous high-resolution mapping of multiple quantitative trait loci in rats: the NIH heterogeneous stock.Arthritis severity locus Cia4 is an early regulator of IL-6, IL-1β, and NF-κB activators' expression in pristane-induced arthritis.Analyses of synovial tissues from arthritic and protected congenic rat strains reveal a new core set of genes associated with disease severityCold-inducible RNA-binding protein (CIRP) causes sepsis-associated acute lung injury via induction of endoplasmic reticulum stress.Recombinant human MFG-E8 ameliorates colon damage in DSS- and TNBS-induced colitis in mice.The multiple chemokine-binding bovine herpesvirus 1 glycoprotein G (BHV1gG) inhibits polymorphonuclear cell but not monocyte migration into inflammatory sites.The GTPase Rac regulates the proliferation and invasion of fibroblast-like synoviocytes from rheumatoid arthritis patients.The non-major histocompatibility complex quantitative trait locus Cia10 contains a major arthritis gene and regulates disease severity, pannus formation, and joint damage.A cold-inducible RNA-binding protein (CIRP)-derived peptide attenuates inflammation and organ injury in septic mice.Short-term low-magnesium diet reduces autoimmune arthritis severity and synovial tissue gene expression.The arthritis severity quantitative trait locus Cia7 regulates neutrophil migration into inflammatory sitesThe arthritis severity quantitative trait loci Cia4 and Cia6 regulate neutrophil migration into inflammatory sites and levels of TNF-alpha and nitric oxideThe non-MHC quantitative trait locus Cia5 contains three major arthritis genes that differentially regulate disease severity, pannus formation, and joint damage in collagen- and pristane-induced arthritisCia27 is a novel non-MHC arthritis severity locus on rat chromosome 10 syntenic to the rheumatoid arthritis 17q22-q25 locusWhat'S New in SHOCK, June 2017?Huntingtin-interacting protein 1 (HIP1) regulates arthritis severity and synovial fibroblast invasiveness by altering PDGFR and Rac1 signallingExtracellular CIRP (eCIRP) and inflammation
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Max Brenner
@ast
Max Brenner
@en
Max Brenner
@es
Max Brenner
@nl
type
label
Max Brenner
@ast
Max Brenner
@en
Max Brenner
@es
Max Brenner
@nl
prefLabel
Max Brenner
@ast
Max Brenner
@en
Max Brenner
@es
Max Brenner
@nl
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P1153
8786647700
P31
P496
0000-0002-8010-148X