about
IVIG regulates the survival of human but not mouse neutrophils.Fc-Galactosylation of Human Immunoglobulin Gamma Isotypes Improves C1q Binding and Enhances Complement-Dependent CytotoxicityRituximab induces sustained reduction of pathogenic B cells in patients with peripheral nervous system autoimmunity.Deregulated Fcγ receptor expression in patients with CIDP.Sialylation of IgG Fc domain impairs complement-dependent cytotoxicity.Efficacy of Intravenous Immunoglobulin in Neurological Diseases.Protection from experimental autoimmune encephalomyelitis by polyclonal IgG requires adjuvant-induced inflammation.Terminal complement activation is increased and associated with disease severity in CIDPFc glycan-modulated immunoglobulin G effector functions.Persistent KSHV Infection Increases EBV-Associated Tumor Formation In Vivo via Enhanced EBV Lytic Gene Expression.Regulation of antibody effector functions through IgG Fc N-glycosylation.The autophagy machinery restrains iNKT cell activation through CD1D1 internalization.ATG-dependent phagocytosis in dendritic cells drives myelin-specific CD4+ T cell pathogenicity during CNS inflammation.Interleukins 12 and 15 induce cytotoxicity and early NK-cell differentiation in type 3 innate lymphoid cellsGeneration of IgG-Fc Glycovariants Using Recombinant Glycosidases and GlycosyltransferasesIVIG efficacy in CIDP patients is not associated with terminal complement inhibition
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description
researcher
@en
wetenschapper
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հետազոտող
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name
Isaak Quast
@ast
Isaak Quast
@en
Isaak Quast
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Isaak Quast
@nl
type
label
Isaak Quast
@ast
Isaak Quast
@en
Isaak Quast
@es
Isaak Quast
@nl
prefLabel
Isaak Quast
@ast
Isaak Quast
@en
Isaak Quast
@es
Isaak Quast
@nl
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P1153
55183902000
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P2798
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P496
0000-0001-6517-8537