Increased expression of sFlt-1 in in vivo and in vitro models of human placental hypoxia is mediated by HIF-1
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Placental origins of adverse pregnancy outcomes: potential molecular targets: an Executive Workshop Summary of the Eunice Kennedy Shriver National Institute of Child Health and Human DevelopmentA lesson for cancer research: placental microarray gene analysis in preeclampsiaIdentification of novel therapeutic targets in microdissected clear cell ovarian cancersPlacental Origins of Chronic Disease.Relationships between TGFbeta proteins and oxygen concentrations inside the first trimester human gestational sacMolecular mechanisms of preeclampsia.Effects of hypoxia-inducible factor-1alpha overexpression in pregnant mice: possible implications for preeclampsia and intrauterine growth restrictionPreeclampsia, of mice and women.Maternal circulating levels of activin A, inhibin A, sFlt-1 and endoglin at parturition in normal pregnancy and pre-eclampsia.Enhancement of the HIF-1α/15-LO/15-HETE axis promotes hypoxia-induced endothelial proliferation in preeclamptic pregnancy.Inflammation-induced fetal growth restriction in rats is associated with increased placental HIF-1α accumulation.Do anti-angiogenic or angiogenic factors contribute to the protection of birth weight at high altitude afforded by Andean ancestry?Recombinant vascular endothelial growth factor 121 infusion lowers blood pressure and improves renal function in rats with placentalischemia-induced hypertension.Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies.Oxidative injury of the pulmonary circulation in the perinatal period: Short- and long-term consequences for the human cardiopulmonary system.Maternal and fetoplacental hypoxia do not alter circulating angiogenic growth effectors during human pregnancy.Beyond oxygen: complex regulation and activity of hypoxia inducible factors in pregnancy.Genome-wide hypermethylation coupled with promoter hypomethylation in the chorioamniotic membranes of early onset pre-eclampsia.Maternally sequestered therapeutic polypeptides - a new approach for the management of preeclampsiaSera from preeclampsia patients elicit symptoms of human disease in mice and provide a basis for an in vitro predictive assayRecent insights into the pathophysiology of preeclampsia.Regulation of sFlt-1 and VEGF secretion by adenosine under hypoxic conditions in rat placental villous explants.Endocrine disruptors, environmental oxygen, epigenetics and pregnancyPro-inflammatory profile of preeclamptic placental mesenchymal stromal cells: new insights into the etiopathogenesis of preeclampsia.Magnetic resonance imaging detects placental hypoxia and acidosis in mouse models of perturbed pregnancies.Landscape of transcriptional deregulations in the preeclamptic placenta.Induction of heme oxygenase 1 attenuates placental ischemia-induced hypertension.Attenuation of VEGFR-2 expression by sFlt-1 and low oxygen in human placentaGlyceryl trinitrate inhibits hypoxia-induced release of soluble fms-like tyrosine kinase-1 and endoglin from placental tissuesSevere early-onset preeclampsia is not associated with a change in placental catechol O-methyltransferase (COMT) expression.Endometrial VEGF induces placental sFLT1 and leads to pregnancy complicationsEffects of heme oxygenase-1 on induction and development of chemically induced squamous cell carcinoma in mice.Regulation of soluble fms-like tyrosine kinase-1 production in response to placental ischemia/hypoxia: role of angiotensin II.Hypoxia-inducible factors and the prevention of acute organ injury.Heme Oxygenase-1 Attenuates Hypoxia-Induced sFlt-1 and Oxidative Stress in Placental Villi through Its Metabolic Products CO and Bilirubin.Placental Features of Late-Onset Adverse Pregnancy Outcome.Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling.Nitric oxide generation affects pro- and anti-angiogenic growth factor expression in primary human trophoblast.Placental metabolic reprogramming: do changes in the mix of energy-generating substrates modulate fetal growth?Hypoxia-Mediated Soluble Fms-Like Tyrosine Kinase 1 Increase Is Not Attenuated in Interleukin 6-Deficient Mice
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Increased expression of sFlt-1 in in vivo and in vitro models of human placental hypoxia is mediated by HIF-1
description
article
@en
im Oktober 2006 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в жовтні 2006
@uk
name
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@en
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@nl
type
label
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@en
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@nl
prefLabel
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@en
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@nl
P2093
P2860
P1476
Increased expression of sFlt-1 ...... l hypoxia is mediated by HIF-1
@en
P2093
Ariel Many
John Kingdom
Nima Soleymanlou
Stacy Zamudio
P2860
P304
P356
10.1152/AJPREGU.00794.2005
P407
P577
2006-04-20T00:00:00Z