about
C7orf30 is necessary for biogenesis of the large subunit of the mitochondrial ribosomeAlternative translation initiation augments the human mitochondrial proteomeMRM2 and MRM3 are involved in biogenesis of the large subunit of the mitochondrial ribosome.Near-complete elimination of mutant mtDNA by iterative or dynamic dose-controlled treatment with mtZFNs.Mitochondrially targeted ZFNs for selective degradation of pathogenic mitochondrial genomes bearing large-scale deletions or point mutations.C7orf30 is necessary for biogenesis of the large subunit of the mitochondrial ribosomeEngineered mtZFNs for Manipulation of Human Mitochondrial DNA Heteroplasmy.Mitochondrial Genome Engineering: The Revolution May Not Be CRISPR-Ized.Heterozygous SSBP1 start loss mutation co-segregates with hearing loss and the m.1555A>G mtDNA variant in a large multigenerational family.NADH Shuttling Couples Cytosolic Reductive Carboxylation of Glutamine with Glycolysis in Cells with Mitochondrial Dysfunction.Linear mitochondrial DNA is rapidly degraded by components of the replication machinery.Genome editing in mitochondria corrects a pathogenic mtDNA mutation in vivoDelivery of mtZFNs into Early Mouse EmbryosEnhanced Manipulation of Human Mitochondrial DNA Heteroplasmy In Vitro Using Tunable mtZFN TechnologyMitochondrially-targeted APOBEC1 is a potent mtDNA mutator affecting mitochondrial function and organismal fitness in DrosophilaEnergetic costs of cellular and therapeutic control of stochastic mitochondrial DNA populations
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