about
Modeling ALS with motor neurons derived from human induced pluripotent stem cellsMechanistic insights into reprogramming to induced pluripotency.X chromosome reactivation dynamics reveal stages of reprogramming to pluripotencyC9orf72 BAC Transgenic Mice Display Typical Pathologic Features of ALS/FTDALS disrupts spinal motor neuron maturation and aging pathways within gene co-expression networksStage-specific regulation of reprogramming to induced pluripotent stem cells by Wnt signaling and T cell factor proteins.C9orf72 is required for proper macrophage and microglial function in mice.The transcription factor NFIA controls the onset of gliogenesis in the developing spinal cord.Modeling Psychomotor Retardation using iPSCs from MCT8-Deficient Patients Indicates a Prominent Role for the Blood-Brain Barrier.The Library of Integrated Network-Based Cellular Signatures NIH Program: System-Level Cataloging of Human Cells Response to Perturbations.Human iPSC-Derived Endothelial Cells and Microengineered Organ-Chip Enhance Neuronal Development.Restoring mitofusin balance prevents axonal degeneration in a Charcot-Marie-Tooth type 2A modeliPSC modeling of young-onset Parkinson's disease reveals a molecular signature of disease and novel therapeutic candidates
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Ritchie Ho
@ast
Ritchie Ho
@en
Ritchie Ho
@es
Ritchie Ho
@nl
type
label
Ritchie Ho
@ast
Ritchie Ho
@en
Ritchie Ho
@es
Ritchie Ho
@nl
prefLabel
Ritchie Ho
@ast
Ritchie Ho
@en
Ritchie Ho
@es
Ritchie Ho
@nl
P106
P1153
15135580300
P31
P496
0000-0003-1496-4436