Mouse hepatitis coronavirus replication induces host translational shutoff and mRNA decay, with concomitant formation of stress granules and processing bodies
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The HTLV-1 Tax protein inhibits formation of stress granules by interacting with histone deacetylase 6Who Regulates Whom? An Overview of RNA Granules and Viral InfectionsRegulation of Stress Responses and Translational Control by CoronavirusAlterations in stress granule dynamics driven by TDP-43 and FUS: a link to pathological inclusions in ALS?Stress granules in the viral replication cycleCoronavirus gene 7 counteracts host defenses and modulates virus virulenceCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionDissection of the influenza A virus endocytic routes reveals macropinocytosis as an alternative entry pathwayHepatitis C virus (HCV) induces formation of stress granules whose proteins regulate HCV RNA replication and virus assembly and egress.Poliovirus unlinks TIA1 aggregation and mRNA stress granule formation.Modulation of stress granules and P bodies during dicistrovirus infection.Respiratory syncytial virus induces host RNA stress granules to facilitate viral replicationStable formation of compositionally unique stress granules in virus-infected cells.Improved microarray gene expression profiling of virus-infected cells after removal of viral RNAType I interferon receptor-independent and -dependent host transcriptional responses to mouse hepatitis coronavirus infection in vivoViral and Cellular mRNA Translation in Coronavirus-Infected Cells.The coronavirus nucleocapsid is a multifunctional protein.Proteomics analysis of differentially expressed proteins in chicken trachea and kidney after infection with the highly virulent and attenuated coronavirus infectious bronchitis virus in vivo.Roles of the respiratory syncytial virus trailer region: effects of mutations on genome production and stress granule formationBUHO: a MATLAB script for the study of stress granules and processing bodies by high-throughput image analysis.Relationship of GW/P-bodies with stress granules.Dynamic Interaction of Stress Granules, DDX3X, and IKK-α Mediates Multiple Functions in Hepatitis C Virus InfectionA novel feedback loop regulates the response to endoplasmic reticulum stress via the cooperation of cytoplasmic splicing and mRNA translationGenetic evidence of a long-range RNA-RNA interaction between the genomic 5' untranslated region and the nonstructural protein 1 coding region in murine and bovine coronavirusesIFN-β-inducing, unusual viral RNA species produced by paramyxovirus infection accumulated into distinct cytoplasmic structures in an RNA-type-dependent mannerMiddle East Respiratory Coronavirus Accessory Protein 4a Inhibits PKR-Mediated Antiviral Stress ResponsesLung epithelial cells have virus-specific and shared gene expression responses to infection by diverse respiratory viruses.Induction of stress granule-like structures in vesicular stomatitis virus-infected cellsDiversion of stress granules and P-bodies during viral infection.Regulation of stress granules and P-bodies during RNA virus infection.Cytoplasmic RNA Granules and Viral Infection.Translationally repressed mRNA transiently cycles through stress granules during stress.Poliovirus infection induces the co-localization of cellular protein SRp20 with TIA-1, a cytoplasmic stress granule protein.Severe acute respiratory syndrome coronavirus triggers apoptosis via protein kinase R but is resistant to its antiviral activityReselection of a genomic upstream open reading frame in mouse hepatitis coronavirus 5'-untranslated-region mutantsPorcine Reproductive and Respiratory Syndrome Virus Infection Induces Stress Granule Formation Depending on Protein Kinase R-like Endoplasmic Reticulum Kinase (PERK) in MARC-145 Cells.RNA granules: the good, the bad and the ugly.Regulation of stress granules in virus systemsTinkering with translation: protein synthesis in virus-infected cells.RNA metabolism in ALS: when normal processes become pathological.
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Mouse hepatitis coronavirus replication induces host translational shutoff and mRNA decay, with concomitant formation of stress granules and processing bodies
description
im September 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 08 May 2007
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована у вересні 2007
@uk
name
Mouse hepatitis coronavirus re ...... granules and processing bodies
@en
Mouse hepatitis coronavirus re ...... granules and processing bodies
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type
label
Mouse hepatitis coronavirus re ...... granules and processing bodies
@en
Mouse hepatitis coronavirus re ...... granules and processing bodies
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prefLabel
Mouse hepatitis coronavirus re ...... granules and processing bodies
@en
Mouse hepatitis coronavirus re ...... granules and processing bodies
@nl
P50
P1476
Mouse hepatitis coronavirus re ...... granules and processing bodies
@en
P2093
Cornelis A M de Haan
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P304
P356
10.1111/J.1462-5822.2007.00951.X
P577
2007-05-08T00:00:00Z