about
Considerations for pre-clinical models and clinical trials of pluripotent stem cell-derived cardiomyocytesCorrection of human phospholamban R14del mutation associated with cardiomyopathy using targeted nucleases and combination therapy.Small molecule-mediated directed differentiation of human embryonic stem cells toward ventricular cardiomyocytes.Growth factor-induced mobilization of cardiac progenitor cells reduces the risk of arrhythmias, in a rat model of chronic myocardial infarction.Effectiveness of gene delivery systems for pluripotent and differentiated cells.Long-term treatment with ivabradine in post-myocardial infarcted rats counteracts f-channel overexpression.Molecular and functional evidence of HCN4 and caveolin-3 interaction during cardiomyocyte differentiation from human embryonic stem cells.Electrophysiologic changes in heart failure: focus on pacemaker channels.Modeling susceptibility to drug-induced long QT with a panel of subject-specific induced pluripotent stem cells.Molecular basis of funny current (If) in normal and failing human heart.Differential functional effects of two 5-HT4 receptor isoforms in adult cardiomyocytes.The direct stimulation of Gi proteins by neuropeptide Y (NPY) in the rat left ventricle.Late sodium current inhibition reverses electromechanical dysfunction in human hypertrophic cardiomyopathy.Gene Transfer in Cardiomyocytes Derived from ES and iPS Cells.Chronic atrial fibrillation alters the functional properties of If in the human atrium.Expression of the hyperpolarization-activated current, I(f), in cultured adult rat ventricular cardiomyocytes and its modulation by hypertrophic factors.Functional remodeling in post-myocardial infarcted rats: focus on beta-adrenoceptor subtypes.Functional Human Beige Adipocytes From Induced Pluripotent Stem Cells.Prenatal exposure to carbon monoxide delays postnatal cardiac maturation.Genomic correction of familial cardiomyopathy in human engineered cardiac tissues.Heart rate reduction with ivabradine prevents the global phenotype of left ventricular remodelingResponse to Letter Regarding Article, “Late Sodium Current Inhibition Reverses Electromechanical Dysfunction in Human Hypertrophic Cardiomyopathy”Functional and transcriptomic insights into pathogenesis of R9C phospholamban mutation using human induced pluripotent stem cell-derived cardiomyocytes
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Francesca Stillitano
@ast
Francesca Stillitano
@en
Francesca Stillitano
@es
Francesca Stillitano
@nl
type
label
Francesca Stillitano
@ast
Francesca Stillitano
@en
Francesca Stillitano
@es
Francesca Stillitano
@nl
prefLabel
Francesca Stillitano
@ast
Francesca Stillitano
@en
Francesca Stillitano
@es
Francesca Stillitano
@nl
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P1153
8549436000
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P31
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0000-0003-1898-5058