about
Effects of the thromboxane receptor agonist U46619 and endothelin-1 on large and small airways.Ether lipids in the cell membrane of Mycoplasma fermentans.A disintegrin and metalloproteinase 17 (ADAM17) mediates inflammation-induced shedding of syndecan-1 and -4 by lung epithelial cellsRecurrent recruitment manoeuvres improve lung mechanics and minimize lung injury during mechanical ventilation of healthy mice.Ventilation-induced lung injury and mechanotransduction: stretching it too far?Quinidine, but not eicosanoid antagonists or dexamethasone, protect the gut from platelet activating factor-induced vasoconstriction, edema and paralysis.The physiological consequences of glutathione variations.Quantification of apoptotic and lytic cell death by video microscopy in combination with artificial neural networks.ADAM-family metalloproteinases in lung inflammation: potential therapeutic targets.Requirements for leukocyte transmigration via the transmembrane chemokine CX3CL1.DNA microarray analysis of gene expression in alveolar epithelial cells in response to TNFalpha, LPS, and cyclic stretch.The early allergic response in small airways of human precision-cut lung slices.Shear Stress Counteracts Endothelial CX3CL1 Induction and Monocytic Cell Adhesion.Platelet-activating factor reduces endothelial nitric oxide production: role of acid sphingomyelinase.Differential effects of the mixed ET(A)/ET(B)-receptor antagonist bosentan on endothelin-induced bronchoconstriction, vasoconstriction and prostacyclin release.Airway relaxant and anti-inflammatory properties of a PDE4 inhibitor with low affinity for the high-affinity rolipram binding site.Platelet-activating factor-induced pulmonary edema is partly mediated by prostaglandin E(2), E-prostanoid 3-receptors, and potassium channels.Altered pulmonary vascular reactivity in mice with excessive erythrocytosis.PAF-mediated pulmonary edema: a new role for acid sphingomyelinase and ceramide.Pressor responses to platelet-activating factor and thromboxane are mediated by Rho-kinase.Sphingolipids in the lungs.Ex vivo lung function measurements in precision-cut lung slices (PCLS) from chemical allergen-sensitized mice represent a suitable alternative to in vivo studies.Measuring the weight of the isolated perfused rat lung during negative pressure ventilation.Mechanical Stress and the Induction of Lung Fibrosis via the Midkine Signaling Pathway.Angiotensin-converting enzyme 2 protects from severe acute lung failure.Modulation of bacterial growth by tumor necrosis factor-alpha in vitro and in vivo.Thrombin stimulates albumin transcytosis in lung microvascular endothelial cells via activation of acid sphingomyelinase.Taking a peep at the upper airways.Effect of surfactant on ventilation-induced mediator release in isolated perfused mouse lungs.Exogenous surfactant reduces ventilator-induced decompartmentalization of tumor necrosis factor alpha in absence of positive end-expiratory pressure.Difficulties in modelling ARDS (2017 Grover Conference Series).Reperfusion-induced gene expression profiles in rat lung transplantation.Reduced rather than enhanced cholinergic airway constriction in mice with ablation of the large conductance Ca2+-activated K+ channelVariable Tidal Volumes Improve Lung Protective Ventilation Strategies in Experimental Lung InjuryTolerance against tumor necrosis factor alpha (TNF)-induced hepatotoxicity in mice: the role of nitric oxideInterleukin-1 and nitric oxide protect against tumor necrosis factor alpha-induced liver injury through distinct pathwaysAn improved setup for the isolated perfused rat lungPhosphodiesterase inhibitors prevent endothelin-1-induced vasoconstriction, bronchoconstriction, and thromboxane release in perfused rat lungICE-protease inhibitors block murine liver injury and apoptosis caused by CD95 or by TNF-alphaDifferent ventilation strategies affect lung function but do not increase tumor necrosis factor-alpha and prostacyclin production in lavaged rat lungs in vivo
P50
Q30915305-3552609A-2791-47C6-BF20-E3D6DAEBC5A5Q30933403-CD795589-8237-4E18-A52B-051C4E0DCAD0Q33581409-75468C72-8B7F-4543-8AF6-ABE257B4DB34Q34026135-924E1AE2-D35B-4AE8-9CA2-CE6C8BF5D747Q34594370-CA8C0521-B8BC-4B1A-9BA2-058160E5A028Q35202866-54157F11-4C60-4FE2-AEC7-F78D1619EFEBQ35796419-65C7399C-6DD8-4E67-B5AD-5B44554DEE1EQ36895180-C91854BC-4BAA-4976-8729-22B7A2FE629DQ38283936-4DF7FDCB-E596-49D4-81C4-752DF2860ACFQ39691199-7200B6A1-A979-45C9-B444-6064B98C3F41Q40509762-1CD1C4D0-9D4F-4708-A53B-FCD8733696DEQ40582508-F8983173-B383-4BB7-9D7B-F98F12C6C726Q42201674-29582098-1292-428D-9207-1874F78D7463Q43238385-446FC16F-1DD4-4DD9-BF29-8AF19E7D7288Q43506529-5A992762-6369-4709-9CE8-099F2D59E4B2Q43937570-D80BAF1D-48A5-4FD4-A73B-36C0CD5C6F03Q44120912-443A62B5-F574-4553-B1B9-9798ECC67175Q44710496-1AE1259C-0D4B-4EEB-91BD-9C050893D653Q44712899-7952223A-1EA5-4F43-BAD2-CE35CB0C0EB6Q44830080-BF1A00C6-4973-492C-9C9D-75BB7C5ACEDAQ44965785-499DE143-E874-45E5-93C1-839F190BDF70Q46384774-B7BABE6B-7116-47F1-AE9F-A4F8F07E3A1CQ47353060-063D2B76-DCC4-419C-8E5D-525310806995Q47709378-53F3600D-42B7-448B-85C2-DC4DD2CBFE22Q47768368-010755B8-6FC5-46DE-AA2E-9E2BB2EB323DQ48956290-600BC504-D317-4342-B520-452A6AF247D5Q51549184-934B2923-FDBC-42A8-B4A4-F40C83FFF533Q51646797-55FF7586-AB4F-4DB2-8FA9-0C7FB3B17D69Q51699199-0FC290D8-9ECA-4E53-BCF5-176271510D2CQ51706662-D480A51A-AB94-42B1-8D73-8F20B13CF2BEQ52665127-96E96810-4319-483E-818C-AE96A767F18CQ54649150-FA992F49-4A5F-47AB-B130-7992AA9618B1Q57987892-C75C8674-7B17-4D24-9790-0CD20765800CQ58994236-00EB5E41-D6EE-4179-B593-5535CF9390E8Q70991037-0F9F5E1B-B0F3-40DD-905C-CE57B114476CQ71565032-B842F4C0-9C90-4E70-A1A5-A1D189E05F22Q72063037-1B5CA062-48FC-4F03-B4AA-9F859DF4E65EQ73154907-012C1602-DC5E-4049-9C0D-E0092E73DB36Q73203922-74DEEC4C-00A4-4F64-908B-64C4ADF3C78BQ73273914-E61CEF64-218B-42C3-8428-4BF957E5040B
P50
description
researcher
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wetenschapper
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հետազոտող
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Stefan Uhlig
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P1053
A-7290-2014
P106
P21
P31
P496
0000-0003-2332-1280