about
RhoE deficiency produces postnatal lethality, profound motor deficits and neurodevelopmental delay in miceMotoneuron survival is enhanced in the absence of neuromuscular junction formation in embryosComparative transcriptomics reveals RhoE as a novel regulator of actin dynamics in bone-resorbing osteoclasts.Differential expression of alpha-CGRP and beta-CGRP genes within hypoglossal motoneurons in response to axotomy.Developmental regulation of glucose transporters GLUT3, GLUT4 and GLUT8 in the mouse cerebellar cortex.RhoE is spatiotemporally regulated in the postnatal mouse CNS.Differential expression of glucose transporter GLUT8 during mouse spermatogenesis.Neuronal polarization is impaired in mice lacking RhoE expression.Expression of the genes for alpha-type and beta-type calcitonin gene-related peptide during postnatal rat brain development.The two mature transcripts of the chick calcitonin gene are expressed within the central nervous system during embryogenesis.Glucose Transporter GLUT8 mRNA Expression in Intestinal Caco-2 Cells is Regulated by Growth and MetabolismNGF-induced motoneuron cell death depends on the genetic background and motoneuron sub-typeSoluble TNF receptors partially protect injured motoneurons in the postnatal CNSUnilateral agenesis of the thyrohyoid muscle in a dogAnatomical variations in the cephalic and omobrachial veins in the dogAnomalous insertion of the splenius muscle in a dogGlucose transporters GLUT4 and GLUT8 are upregulated after facial nerve axotomy in adult mice
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P50
description
researcher ORCID ID = 0000-0002-7356-2341
@en
wetenschapper
@nl
name
José Terrado
@ast
José Terrado
@en
José Terrado
@es
José Terrado
@nl
type
label
José Terrado
@ast
José Terrado
@en
José Terrado
@es
José Terrado
@nl
prefLabel
José Terrado
@ast
José Terrado
@en
José Terrado
@es
José Terrado
@nl
P106
P1153
6603654125
P21
P31
P496
0000-0002-7356-2341