BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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BORIS: a key regulator of cancer stemness

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P1476

BORIS: a key regulator of cancer stemness

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P2093

Sara Soltanian

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P2860

The testis-specific factor CTCFL cooperates with the protein methyltransferase PRMT7 in H19 imprinting control region methylation

Translating the Histone Code

Intronic promoter drives the BORIS-regulated expression of FerT in colon carcinoma cells

PRMT7, a new protein arginine methyltransferase that synthesizes symmetric dimethylarginine

BAT3 and SET1A form a complex with CTCFL/BORIS to modulate H3K4 histone dimethylation and gene expression

Reciprocal binding of CTCF and BORIS to the NY-ESO-1 promoter coincides with derepression of this cancer-testis gene in lung cancer cells

DNA methyltransferase 1 and 3B activate BAG-1 expression via recruitment of CTCFL/BORIS and modulation of promoter histone methylation

BORIS, a novel male germ-line-specific protein associated with epigenetic reprogramming events, shares the same 11-zinc-finger domain with CTCF, the insulator protein involved in reading imprinting marks in the soma.

A distinct "side population" of cells with high drug efflux capacity in human tumor cells

DNA methylation-dependent regulation of BORIS/CTCFL expression in ovarian cancer

P2888

s12935-018-0650-8

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154

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scholarly article

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10.1186/S12935-018-0650-8

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P407

English

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P478

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P50

Hesam Dehghani

P577

2018-01-01T00:00:00Z

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P6179

1107429331

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P698

30323717

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P921

cancer research

P932

6173857

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