KLF10gene expression is associated with high fetal hemoglobin levels and with response to hydroxyurea treatment in β-hemoglobinopathy patients - Wikidata - awgldk - TriplyDB

KLF10gene expression is associated with high fetal hemoglobin levels and with response to hydroxyurea treatment in β-hemoglobinopathy patients

KLF10gene expression is associated with high fetal hemoglobin levels and with response to hydroxyurea treatment in β-hemoglobinopathy patients

http://www.wikidata.org/entity/Q57618969

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Minireview: Prognostic factors and the response to hydroxurea treatment in sickle cell disease.Frequency of Hospitalizations for Pain and Association With Altered Brain Network Connectivity in Sickle Cell Disease.Advances in technologies for screening and diagnosis of hemoglobinopathies.Recent trends for novel options in experimental biological therapy of β-thalassemia.Individualizing fetal hemoglobin augmenting therapy for β-type hemoglobinopathies patients.Recent trends in the gene therapy of β-thalassemia.Preconception carrier screening and prenatal diagnosis in thalassemia and hemoglobinopathies: challenges and future perspectives.An Expert Review of Pharmacogenomics of Sickle Cell Disease Therapeutics: Not Yet Ready for Global Precision Medicine.Study on Hydroxyurea Response in Hemoglobinopathies Patients Using Genetic Markers and Liquid Erythroid Cultures.Evaluation of the Relationship Between Hb F Levels and Nucleated Red Blood Cells with Morbidity in Non Transfusion-Dependent Thalassemia Patients.Whole transcriptome analysis of human erythropoietic cells during ontogenesis suggests a role of VEGFA gene as modulator of fetal hemoglobin and pharmacogenomic biomarker of treatment response to hydroxyurea in β-type hemoglobinopathy patients Pharmacogenomics of sickle cell disease: steps toward personalized medicine.Pharmacological and molecular approaches for the treatment of β-hemoglobin disorders.Genomic variation in the MAP3K5 gene is associated with β-thalassemia disease severity and hydroxyurea treatment efficacy.Pharmacogenomics education and research at the Department of Pharmacy, University of Patras, Greece.First description of the rs45496295 polymorphism of the C/EBPE gene in β-thalassemia intermedia patients.Correlation of SIN3A genomic variants with β-hemoglobinopathies disease severity and hydroxyurea treatment efficacy.Genomic variants in the ASS1 gene, involved in the nitric oxide biosynthesis and signaling pathway, predict hydroxyurea treatment efficacy in compound sickle cell disease/β-thalassemia patients.Functional analysis of a novel KLF1 gene promoter variation associated with hereditary persistence of fetal hemoglobin.

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KLF10gene expression is associated with high fetal hemoglobin levels and with response to hydroxyurea treatment in β-hemoglobinopathy patients

http://www.wikidata.org/entity/Q57618969

description

article

@en

wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2012

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name

KLF10gene expression is associ ...... in β-hemoglobinopathy patients

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KLF10gene expression is associ ...... in β-hemoglobinopathy patients

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type

label

KLF10gene expression is associ ...... in β-hemoglobinopathy patients

@en

KLF10gene expression is associ ...... in β-hemoglobinopathy patients

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prefLabel

KLF10gene expression is associ ...... in β-hemoglobinopathy patients

@en

KLF10gene expression is associ ...... in β-hemoglobinopathy patients

@nl

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Pharmacogenomics

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KLF10gene expression is associ ...... in β-hemoglobinopathy patients

@en

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Adamantia Papachatzopoulou

http://www.w3.org/2001/XMLSchema#string

Alexander E Felice

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Alexandra Kourakli

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Carsten Lederer

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Christina Lappa-Manakou

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Christina Tafrali

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Eleana F Stavrou

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Frank G Grosveld

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George P Patrinos

http://www.w3.org/2001/XMLSchema#string

Jun Hou

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P2860

Genome annotation of a 1.5 Mb region of human chromosome 6q23 encompassing a quantitative trait locus for fetal hemoglobin expression in adults

Haploinsufficiency for the erythroid transcription factor KLF1 causes hereditary persistence of fetal hemoglobin

Krüppel-like Factor 4 activates HBG gene expression in primary erythroid cells

A conserved alpha-helical motif mediates the interaction of Sp1-like transcriptional repressors with the corepressor mSin3A

Transcription factors KLF1 and KLF2 positively regulate embryonic and fetal beta-globin genes through direct promoter binding

Genome-wide association study shows BCL11A associated with persistent fetal hemoglobin and amelioration of the phenotype of beta-thalassemia

Systematic RNAi studies on the role of Sp/KLF factors in globin gene expression and erythroid differentiation

Java Treeview--extensible visualization of microarray data

Open source clustering software

Synergistic enhancement of globin gene expression by activator protein-1-like proteins.

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1487-1500

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scholarly article

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10.2217/PGS.12.125

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13

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13

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Wilfred F J van IJcken

Marianthi Georgitsi

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2012-10-01T00:00:00Z

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23057549

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