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De novo formation of left-right asymmetry by posterior tilt of nodal ciliaEstablishment of vertebrate left-right asymmetryOrigin of body axes in the mouse embryoLeft-right asymmetric expression of the TGF beta-family member lefty in mouse embryosNodal antagonists regulate formation of the anteroposterior axis of the mouse embryoNotch signaling regulates left-right asymmetry determination by inducing Nodal expressionPih1d3 is required for cytoplasmic preassembly of axonemal dynein in mouse spermSitus inversus and ciliary abnormalities: 20 years later, what is the connection?Left-right asymmetry in the level of active Nodal protein produced in the node is translated into left-right asymmetry in the lateral plate of mouse embryos.Translation of anterior-posterior polarity into left-right polarity in the mouse embryo.Origin of cellular asymmetries in the pre-implantation mouse embryo: a hypothesis.Mechanisms of left-right asymmetry and patterning: driver, mediator and responderTwo rotating cilia in the node cavity are sufficient to break left-right symmetry in the mouse embryo.Antagonism between Smad1 and Smad2 signaling determines the site of distal visceral endoderm formation in the mouse embryo.Removal of maternal retinoic acid by embryonic CYP26 is required for correct Nodal expression during early embryonic patterningCell fate decisions and axis determination in the early mouse embryo.Roles of cilia, fluid flow, and Ca2+ signaling in breaking of left-right symmetry.TGFβ signaling in establishing left-right asymmetry.Role of physical forces in embryonic development.Origin and role of distal visceral endoderm, a group of cells that determines anterior-posterior polarity of the mouse embryo.Absence of Radial Spokes in Mouse Node Cilia Is Required for Rotational Movement but Confers Ultrastructural Instability as a Trade-Off.Planar polarization of node cells determines the rotational axis of node cilia.Cloning of inv, a gene that controls left/right asymmetry and kidney development.In search of Turing in vivo: understanding Nodal and Lefty behavior.Hydrodynamic phase locking in mouse node cilia.Reversal of left-right asymmetry induced by aberrant Nodal signaling in the node of mouse embryos.Haemodynamics determined by a genetic programme govern asymmetric development of the aortic arch.Tbx6 controls left-right asymmetry through regulation of Gdf1.Loss of Fam60a, a Sin3a subunit, results in embryonic lethality and is associated with aberrant methylation at a subset of gene promotersDetermination of left–right patterning of the mouse embryo by artificial nodal flowWnt signalling escapes to ciliaAsymmetric rotational stroke in mouse node cilia during left-right determinationDiversity of left-right symmetry breaking strategy in animalsCiliogenesis-coupled accumulation of IFT-B proteins in a novel cytoplasmic compartmentNodal paralogues underlie distinct mechanisms for visceral left-right asymmetry in reptiles and mammals
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-7196-5948
@en
name
Hiroshi Hamada
@ast
Hiroshi Hamada
@en
Hiroshi Hamada
@es
Hiroshi Hamada
@nl
type
label
Hiroshi Hamada
@ast
Hiroshi Hamada
@en
Hiroshi Hamada
@es
Hiroshi Hamada
@nl
prefLabel
Hiroshi Hamada
@ast
Hiroshi Hamada
@en
Hiroshi Hamada
@es
Hiroshi Hamada
@nl
P106
P31
P496
0000-0002-7196-5948