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Comparison of Experimental Protocols of Physical Exercise for mdx Mice and Duchenne Muscular Dystrophy Patients.Impaired energy metabolism of senescent muscle satellite cells is associated with oxidative modifications of glycolytic enzymes.Oxidative stress-induced proteome alterations target different cellular pathways in human myoblasts.Low intensity training of mdx mice reduces carbonylation and increases expression levels of proteins involved in energy metabolism and muscle contraction.Low-Intensity Training and the C5a Complement Antagonist NOX-D21 Rescue the mdx Phenotype through Modulation of Inflammation.Progressive skeletal muscle weakness in transgenic mice expressing CTG expansions is associated with the activation of the ubiquitin–proteasome pathwayImprovement of Dystrophic Muscle Fragility by Short-Term Voluntary Exercise through Activation of Calcineurin Pathway in mdx Mice
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description
onderzoeker
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researcher ORCID ID = 0000-0002-2333-9336
@en
name
Janek Hyzewicz
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Janek Hyzewicz
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Janek Hyzewicz
@es
Janek Hyzewicz
@nl
type
label
Janek Hyzewicz
@ast
Janek Hyzewicz
@en
Janek Hyzewicz
@es
Janek Hyzewicz
@nl
prefLabel
Janek Hyzewicz
@ast
Janek Hyzewicz
@en
Janek Hyzewicz
@es
Janek Hyzewicz
@nl
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0000-0002-2333-9336