MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene - Wikidata - awgldk - TriplyDB

MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene

MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene

http://www.wikidata.org/entity/Q57690359

about

Tumour microvesicles contain retrotransposon elements and amplified oncogene sequences Differential ability of Tribbles family members to promote degradation of C/EBPalpha and induce acute myelogenous leukemia An integrative framework for the identification of double minute chromosomes using next generation sequencing data.Chromosomal minimal critical regions in therapy-related leukemia appear different from those of de novo leukemia by high-resolution aCGH.A common copy-number breakpoint of ERBB2 amplification in breast cancer colocalizes with a complex block of segmental duplications.Genomic organization and evolution of double minutes/homogeneously staining regions with MYC amplification in human cancer.Gain of MYC underlies recurrent trisomy of the MYC chromosome in acute promyelocytic leukemia Amplicon rearrangements during the extrachromosomal and intrachromosomal amplification process in a glioma Constitutive ERK1/2 activation contributes to production of double minute chromosomes in tumour cells Myc roles in hematopoiesis and leukemia Novel role for non-homologous end joining in the formation of double minutes in methotrexate-resistant colon cancer cells.Integration of microarray analysis into the clinical diagnosis of hematological malignancies: How much can we improve cytogenetic testing?Cytogenetic, molecular genetic, and clinical characteristics of acute myeloid leukemia with a complex karyotype.Brain tumor microvesicles: insights into intercellular communication in the nervous system.De novo-generated small palindromes are characteristic of amplicon boundary junction of double minutes.Characterization at nucleotide resolution of the homogeneously staining region sites of insertion in two cancer cell lines.Tribbles in disease: Signaling pathways important for cellular function and neoplastic transformation.Tribbles: 'puzzling' regulators of cell signalling.Unfavorable-risk cytogenetics in acute myeloid leukemia.MYC oncogene in myeloid neoplasias.Somatic structural variation and cancer.Long noncoding RNA, CCDC26, controls myeloid leukemia cell growth through regulation of KIT expression The Evolving Genomic Landscape of Barrett's Esophagus and Esophageal Adenocarcinoma.Gene amplification as double minutes or homogeneously staining regions in solid tumors: origin and structure.TRIB1 promotes colorectal cancer cell migration and invasion through activation MMP-2 via FAK/Src and ERK pathways.Molecular characterization of cell-free eccDNAs in human plasma.Genomic amplification of MYC as double minutes in a patient with APL-like leukemia.Impediment of Replication Forks by Long Non-coding RNA Provokes Chromosomal Rearrangements by Error-Prone Restart.The Hidden Genomic and Transcriptomic Plasticity of Giant Marker Chromosomes in Cancer.Frequency of double minute chromosomes and combined cytogenetic abnormalities and their characteristics.Trib2 expression in granulocyte-monocyte progenitors drives a highly drug resistant acute myeloid leukaemia linked to elevated Bcl2.Association of germline or somatic TP53 missense mutation with oncogene amplification in tumors developed in patients with Li-Fraumeni or Li-Fraumeni-like syndrome.Acute myeloid leukemia Acute Myeloid Leukemia

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MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene

http://www.wikidata.org/entity/Q57690359

description

article

@en

im Februar 2006 veröffentlichter wissenschaftlicher Artikel

@de

wetenschappelijk artikel

@nl

наукова стаття, опублікована в лютому 2006

@uk

name

MYC-containing double minutes ...... sion of MYC as the target gene

@en

MYC-containing double minutes ...... sion of MYC as the target gene

@nl

type

label

MYC-containing double minutes ...... sion of MYC as the target gene

@en

MYC-containing double minutes ...... sion of MYC as the target gene

@nl

prefLabel

MYC-containing double minutes ...... sion of MYC as the target gene

@en

MYC-containing double minutes ...... sion of MYC as the target gene

@nl

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Human Molecular Genetics

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MYC-containing double minutes ...... sion of MYC as the target gene

@en

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Angela Mastrorilli

http://www.w3.org/2001/XMLSchema#string

Anna Aventin

http://www.w3.org/2001/XMLSchema#string

Anne Hagemeijer

http://www.w3.org/2001/XMLSchema#string

Arabella Smith

http://www.w3.org/2001/XMLSchema#string

Bertil Johansson

http://www.w3.org/2001/XMLSchema#string

Bodil Strömbeck

http://www.w3.org/2001/XMLSchema#string

Cecilia Surace

http://www.w3.org/2001/XMLSchema#string

Christa Fonatsch

http://www.w3.org/2001/XMLSchema#string

Claudia Schoch

http://www.w3.org/2001/XMLSchema#string

Dominique Mühlematter

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P2860

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New mammalian cellular systems to study mutations introduced at the break site by non-homologous end-joining.

The breakpoint region of the most common isochromosome, i(17q), in human neoplasia is characterized by a complex genomic architecture with large, palindromic, low-copy repeats

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Microarray analysis reveals a major direct role of DNA copy number alteration in the transcriptional program of human breast tumors.

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933-942

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scholarly article

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10.1093/HMG/DDL010

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6

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15

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Clelia Tiziana Storlazzi

Florence Nguyen-Khac

Pietro D'Addabbo

Crescenzio Francesco Minervini

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2006-02-01T00:00:00Z

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16452126

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