Absence of TMPRSS2:ERG fusions and PTEN losses in prostate cancer is associated with a favorable outcome - Wikidata - awgldk - TriplyDB

Absence of TMPRSS2:ERG fusions and PTEN losses in prostate cancer is associated with a favorable outcome

Absence of TMPRSS2:ERG fusions and PTEN losses in prostate cancer is associated with a favorable outcome

http://www.wikidata.org/entity/Q57790493

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Incorporation of tissue-based genomic biomarkers into localized prostate cancer clinics Emerging Role of Genomic Rearrangements in Breast Cancer: Applying Knowledge from Other Cancers PSA and beyond: alternative prostate cancer biomarkers What is the Need for Prostatic Biomarkers in Prostate Cancer Management?Discovery and Classification of Fusion Transcripts in Prostate Cancer and Normal Prostate Tissue Emerging critical role of molecular testing in diagnostic genitourinary pathology Differential cytotoxic activity of a novel palladium-based compound on prostate cell lines, primary prostate epithelial cells and prostate stem cells Overexpression of ERG and Wild-Type PTEN Are Associated with Favorable Clinical Prognosis and Low Biochemical Recurrence in Prostate Cancer Androgen regulation of the TMPRSS2 gene and the effect of a SNP in an androgen response element Molecular characterisation of ERG, ETV1 and PTEN gene loci identifies patients at low and high risk of death from prostate cancer.Recurrent prostate cancer genomic alterations predict response to brachytherapy treatment Loss of PTEN is associated with aggressive behavior in ERG-positive prostate cancer The role of TMPRSS2:ERG in molecular stratification of PCa and its association with tumor aggressiveness: a study in Brazilian patients.Mouse models of prostate cancer: picking the best model for the question.Evidence for molecular differences in prostate cancer between African American and Caucasian men Novel diagnostic biomarkers for prostate cancer.Discovery of non-ETS gene fusions in human prostate cancer using next-generation RNA sequencing.The ETS family of oncogenic transcription factors in solid tumours.MALDI mass spectrometric imaging based identification of clinically relevant signals in prostate cancer using large-scale tissue microarrays.ERG induces epigenetic activation of Tudor domain-containing protein 1 (TDRD1) in ERG rearrangement-positive prostate cancer.Biologic differences between peripheral and transition zone prostate cancer Oncogenic gene fusions in epithelial carcinomas A three-marker FISH panel detects more genetic aberrations of AR, PTEN and TMPRSS2/ERG in castration-resistant or metastatic prostate cancers than in primary prostate tumors.ERG rearrangement is associated with prostate cancer-related death in Chinese prostate cancer patients Loss of PTEN expression is associated with increased risk of recurrence after prostatectomy for clinically localized prostate cancer Multi-transcript profiling in archival diagnostic prostate cancer needle biopsies to evaluate biomarkers in non-surgically treated men.Frequent gene products and molecular pathways altered in prostate cancer- and metastasis-initiating cells and their progenies and novel promising multitargeted therapies.PTEN protein loss and clinical outcome from castration-resistant prostate cancer treated with abiraterone acetate.Oncogenic activation of ERG: A predominant mechanism in prostate cancer.Integrated analysis of the genomic instability of PTEN in clinically insignificant and significant prostate cancer.ETV1, 4 and 5: an oncogenic subfamily of ETS transcription factors.Germ-line sequence variants of PTEN do not have an important role in hereditary and non-hereditary prostate cancer susceptibility.Colorimetric TMPRSS2-ERG Gene Fusion Detection in Prostate Cancer Urinary Samples via Recombinase Polymerase Amplification TMPRSS2-ERG gene fusion is associated with low Gleason scores and not with high-grade morphological features.ERG immunohistochemistry and clinicopathologic characteristics in Korean prostate adenocarcinoma patients.Integrin-linked kinase as a target for ERG-mediated invasive properties in prostate cancer models Molecular evidence that invasive adenocarcinoma can mimic prostatic intraepithelial neoplasia (PIN) and intraductal carcinoma through retrograde glandular colonization.Personalized management in low-risk prostate cancer: the role of biomarkers.Cytoplasmic PTEN protein loss distinguishes intraductal carcinoma of the prostate from high-grade prostatic intraepithelial neoplasia.A Prospective Investigation of PTEN Loss and ERG Expression in Lethal Prostate Cancer.

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Absence of TMPRSS2:ERG fusions and PTEN losses in prostate cancer is associated with a favorable outcome

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Absence of TMPRSS2:ERG fusions ...... iated with a favorable outcome

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Absence of TMPRSS2:ERG fusions ...... iated with a favorable outcome

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Modern Pathology

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Absence of TMPRSS2:ERG fusions ...... iated with a favorable outcome

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Francisco P Fonseca

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Isabela W Cunha

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Maisa Yoshimoto

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Maria Zielenska

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Olga Ludkovski

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Renata A Coudry

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P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Three-color FISH analysis of TMPRSS2/ERG fusions in prostate cancer indicates that genomic microdeletion of chromosome 21 is associated with rearrangement

TMPRSS2-ERG gene fusion causing ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesions

Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer.

FISH analysis of 107 prostate cancers shows that PTEN genomic deletion is associated with poor clinical outcome.

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Role of the TMPRSS2-ERG gene fusion in prostate cancer

Regulation of cell death protease caspase-9 by phosphorylation

Oncomine 3.0: genes, pathways, and networks in a collection of 18,000 cancer gene expression profiles

Frequent overexpression of ETS-related gene-1 (ERG1) in prostate cancer transcriptome.

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