A thymidine kinase-deleted bovine herpesvirus 5 establishes latent infection but reactivates poorly in a sheep model

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Mapping the sites of latency and reactivation by bovine herpesvirus 5 (BoHV-5) and a thymidine kinase-deleted BoHV-5 in lambs

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Q57874562-0EB01488-C7B3-4530-908A-E1F7F78E5D77

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A thymidine kinase-deleted bovine herpesvirus 5 establishes latent infection but reactivates poorly in a sheep model

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wetenschappelijk artikel

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наукова стаття, опублікована в березні 2013

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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A thymidine kinase-deleted bov ...... ivates poorly in a sheep model

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Deniz Anziliero

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Eduardo F. Flores

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Gustavo C. Cadore

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Marcelo Weiss

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Mário Celso S. Brum

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Rudi Weiblen

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The MIQE guidelines: minimum information for publication of quantitative real-time PCR experiments

Replication of herpes simplex virus type 1 within trigeminal ganglia is required for high frequency but not high viral genome copy number latency.

Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.

Genome of bovine herpesvirus 5.

A mutation in the latency-related gene of bovine herpesvirus 1 disrupts the latency reactivation cycle in calves.

Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishment.

Translational compensation of a frameshift mutation affecting herpes simplex virus thymidine kinase is sufficient to permit reactivation from latency.

Expression of extremely low levels of thymidine kinase from an acyclovir-resistant herpes simplex virus mutant supports reactivation from latently infected mouse trigeminal ganglia

Role of herpes simplex virus thymidine kinase expression in viral pathogenesis and latency.

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331-338

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scholarly article

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10.1590/S0100-736X2013000300010

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2013-03-01T00:00:00Z

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A thymidine kinase-deleted bovine herpesvirus 5 establishes latent infection but reactivates poorly in a sheep model

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A thymidine kinase-deleted bovine herpesvirus 5 establishes latent infection but reactivates poorly in a sheep model

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