Expression of Mutated IGHV3-23 Genes in Chronic Lymphocytic Leukemia Identifies a Disease Subset with Peculiar Clinical and Biological Features
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T-cell intracellular antigens in health and diseaseDetection of TP53 dysfunction in chronic lymphocytic leukemia by an in vitro functional assay based on TP53 activation by the non-genotoxic drug Nutlin-3: a proposal for clinical application.Not all IGHV3-21 chronic lymphocytic leukemias are equal: prognostic considerationsA B-Cell Superantigen Induces the Apoptosis of Murine and Human Malignant B Cells.The SIRT1/TP53 axis is activated upon B-cell receptor triggering via miR-132 up-regulation in chronic lymphocytic leukemia cells.Longitudinal copy number, whole exome and targeted deep sequencing of 'good risk' IGHV-mutated CLL patients with progressive diseaseImmunoglobulin gene rearrangements in Chinese and Italian patients with chronic lymphocytic leukemia.Intrinsic and extrinsic factors influencing the clinical course of B-cell chronic lymphocytic leukemia: prognostic markers with pathogenetic relevance.B-cell receptor, clinical course and prognosis in chronic lymphocytic leukaemia: the growing saga of the IGHV3 subgroup gene usage.Treatment of chronic lymphocytic leukemia requires targeting of the protective lymph node environment with novel therapeutic approaches.13q14 deletion size and number of deleted cells both influence prognosis in chronic lymphocytic leukemia.The Individual and Population Genetics of Antibody Immunity.Potential therapeutic role of antagomiR17 for the treatment of chronic lymphocytic leukemia.Heterogeneous nuclear ribonucleoprotein K supports vesicular stomatitis virus replication by regulating cell survival and cellular gene expressionImmunoglobulin gene repertoire in chronic lymphocytic leukemia: insight into antigen selection and microenvironmental interactions.IGHV gene rearrangements as outcome predictors for CLL patients: experience of Ukrainian group.Chronic Lymphocytic Leukemia B-Cell Normal Cellular Counterpart: Clues From a Functional Perspective.Immunoglobulin heavy chain variable region gene repertoire and B-cell receptor stereotypes in Indian patients with chronic lymphocytic leukemia.The miR-17∼92 family regulates the response to Toll-like receptor 9 triggering of CLL cells with unmutated IGHV genes.
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P2860
Expression of Mutated IGHV3-23 Genes in Chronic Lymphocytic Leukemia Identifies a Disease Subset with Peculiar Clinical and Biological Features
description
article
@en
im Januar 2010 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в січні 2010
@uk
name
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@en
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@nl
type
label
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@en
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@nl
prefLabel
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@en
Expression of Mutated IGHV3-23 ...... inical and Biological Features
@nl
P2093
P50
P1476
Expression of mutated IGHV3-23 ...... inical and biological features
@en
P2093
Daniela Marconi
Davide Rossi
Elisa Sozzi
Fabrizio Luciano
Francesca Maria Rossi
Gianluca Gaidano
Ilaria Cattarossi
Luca Laurenti
Massimo Degan
Milena S Nicoloso
P304
P356
10.1158/1078-0432.CCR-09-1638
P407
P50
P577
2010-01-12T00:00:00Z