Two genetic loci independently confer susceptibility to autoimmune gastritis
about
A convenient model of severe, high incidence autoimmune gastritis caused by polyclonal effector T cells and without perturbation of regulatory T cells.Dominant trait linked to chromosome 1 in DBA/2 mice for the resistance to autoimmune gastritis appears in bone marrow cellsCutting edge issues in autoimmune gastritis.Interleukin-21 and tumor necrosis factor-α are critical for the development of autoimmune gastritis in mice.
P2860
Two genetic loci independently confer susceptibility to autoimmune gastritis
description
im August 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 13 August 2007
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в серпні 2007
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name
Two genetic loci independently confer susceptibility to autoimmune gastritis
@en
Two genetic loci independently confer susceptibility to autoimmune gastritis
@nl
type
label
Two genetic loci independently confer susceptibility to autoimmune gastritis
@en
Two genetic loci independently confer susceptibility to autoimmune gastritis
@nl
prefLabel
Two genetic loci independently confer susceptibility to autoimmune gastritis
@en
Two genetic loci independently confer susceptibility to autoimmune gastritis
@nl
P2093
P50
P356
P1476
Two genetic loci independently confer susceptibility to autoimmune gastritis
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P2093
Briony L Gliddon
Desmond K Y Ang
Margaret A Jordan
Wendy E Wilson
P304
P356
10.1093/INTIMM/DXM087
P577
2007-08-13T00:00:00Z