about
A mutational signature reveals alterations underlying deficient homologous recombination repair in breast cancer.Resistance to checkpoint blockade therapy through inactivation of antigen presentation.Scalable whole-exome sequencing of cell-free DNA reveals high concordance with metastatic tumors.The evolutionary landscape of chronic lymphocytic leukemia treated with ibrutinib targeted therapy.Distinct mutational signatures characterize concurrent loss of polymerase proofreading and mismatch repair.Resolving the phylogenetic origin of glioblastoma via multifocal genomic analysis of pre-treatment and treatment-resistant autopsy specimens.Two-dimensional gas chromatography/mass spectrometry, physical property modeling and automated production of component maps to assess the weathering of pollutantsAuthor Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomesPublisher Correction: Molecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomesMolecular subtypes of diffuse large B cell lymphoma are associated with distinct pathogenic mechanisms and outcomesLiquid versus tissue biopsy for detecting acquired resistance and tumor heterogeneity in gastrointestinal cancersWidespread Chromosomal Losses and Mitochondrial DNA Alterations as Genetic Drivers in Hürthle Cell CarcinomaAuthor Correction: Liquid versus tissue biopsy for detecting acquired resistance and tumor heterogeneity in gastrointestinal cancersGrowth dynamics in naturally progressing chronic lymphocytic leukaemiaRNA sequence analysis reveals macroscopic somatic clonal expansion across normal tissues
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0003-2528-1446
@en
name
D Livitz
@ast
D Livitz
@en
D Livitz
@nl
type
label
D Livitz
@ast
D Livitz
@en
D Livitz
@nl
prefLabel
D Livitz
@ast
D Livitz
@en
D Livitz
@nl
P106
P31
P496
0000-0003-2528-1446