about
Chemical and instrumental approaches to treat hyperpigmentation.Modulation of PPARγ provides new insights in a stress induced premature senescence model.Antioxidant activity, lipid peroxidation and skin diseases. What's new.Hypopigmenting agents: an updated review on biological, chemical and clinical aspects.Melanins and melanogenesis: methods, standards, protocols.Pharmacological PPARγ modulation regulates sebogenesis and inflammation in SZ95 human sebocytes.Cystinosin is a melanosomal protein that regulates melanin synthesis.Keratinocyte growth factor down-regulates intracellular ROS production induced by UVB.Ultraviolet A induced modulation of gap junctional intercellular communication by P38 MAPK activation in human keratinocytes.Ferritin light chain down-modulation generates depigmentation in human metastatic melanoma cells by influencing tyrosinase maturation.The activation of PPARγ by 2,4,6-Octatrienoic acid protects human keratinocytes from UVR-induced damages.Correlation between melanogenic and catalase activity in in vitro human melanocytes: a synergic strategy against oxidative stress.Polyunsaturated fatty acids partially reproduce the role of melanocytes in the epidermal melanin unit.Oxidative stress in physical urticarias.Epidermal reconstructs: a new tool to study topical and systemic photoprotective molecules.Time-kinetic study of repigmentation in vitiligo patients by tacrolimus or pimecrolimus.Modulation of reactive oxygen species via ERK and STAT3 dependent signalling are involved in the response of mesothelioma cells to exemestane.Small molecular antioxidants effectively protect from PUVA-induced oxidative stress responses underlying fibroblast senescence and photoaging.Relationship among lipoperoxides, jasmonates and indole-3-acetic acid formation in potato tuber after wounding.UVA-induced modification of catalase charge properties in the epidermis is correlated with the skin phototype.Scavenging effects of terbinafine on free radicals in vitro.Anti-oxidant defence mechanism in vitiliginous skin increases with skin type.Azelaic acid reduced senescence-like phenotype in photo-irradiated human dermal fibroblasts: possible implication of PPARγ.Chemical, Pharmacologic, and Physical Agents Causing HypomelanosesSimultaneous determination of reduced and oxidized glutathione in peripheral blood mononuclear cells by liquid chromatography–electrospray mass spectrometryCerium Oxide Nanoparticles Re-establish Cell Integrity Checkpoints and Apoptosis Competence in Irradiated HaCat Cells via Novel Redox-Independent ActivityRole of fibroblast-derived growth factors in regulating hyperpigmentation of solar lentigo2,4,6-Octatrienoic acid is a novel promoter of melanogenesis and antioxidant defence in normal human melanocytes via PPAR-γ activationAcidic catalase in human skin in vivo: a new marker of permanent damageFree radical oxidation of 15-(S)-hydroxyeicosatetraenoic acid with the Fenton reagent: characterization of an epoxy-alcohol and cytotoxic 4-hydroxy-2E-nonenal from the heptatrienyl radical pathwayInhibitors and Enhancers of MelanogenesisAccelerated features of senescence in cultured type 2 diabetic skin fibroblasts
P50
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P50
description
onderzoeker
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researcher ORCID ID = 0000-0003-2000-006X
@en
name
S Briganti
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Stefania Briganti
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Stefania Briganti
@es
type
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S Briganti
@nl
Stefania Briganti
@en
Stefania Briganti
@es
altLabel
S Briganti
@en
prefLabel
S Briganti
@nl
Stefania Briganti
@en
Stefania Briganti
@es
P106
P1153
6603308206
P21
P31
P496
0000-0003-2000-006X