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IL-15 triggers an antiapoptotic pathway in human intraepithelial lymphocytes that is a potential new target in celiac disease-associated inflammation and lymphomagenesisCeliac disease: an immunological jigsawPresentation and long-term follow-up of refractory celiac disease: comparison of type I with type II.Celiac disease: from oral tolerance to intestinal inflammation, autoimmunity and lymphomagenesis.Low ileal interleukin 10 concentrations are predictive of endoscopic recurrence in patients with Crohn's disease.Gastrointestinal Disorder Associated with Olmesartan Mimics Autoimmune EnteropathyDesigning 3D Mesenchymal Stem Cell Sheets Merging Magnetic and Fluorescent Features: When Cell Sheet Technology Meets Image-Guided Cell Therapy.Reprogramming of CTLs into natural killer-like cells in celiac diseaseThe cytokine interleukin 21: a new player in coeliac disease?Innate T cell responses in human gut.Enteropathy-associated T-cell lymphoma: a review on clinical presentation, diagnosis, therapeutic strategies and perspectives.Refractory celiac disease: from bench to bedside.Lessons from rodent models in celiac disease.Interleukin-15, a master piece in the immunological jigsaw of celiac disease.The role of animal models in unravelling therapeutic targets in coeliac disease.Discovery and characterization of a novel humanized anti-IL-15 antibody and its relevance for the treatment of refractory celiac disease and eosinophilic esophagitis.Human NKG2E is expressed and forms an intracytoplasmic complex with CD94 and DAP12.Small intestinal CD4+ T-cell lymphoma is a heterogenous entity with common pathology features.Olmesartan-associated enteropathy: results of a national survey.Interleukin-15-dependent NKp46+ innate lymphoid cells control intestinal inflammation by recruiting inflammatory monocytes.Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease.Interleukin-15-Dependent T-Cell-like Innate Intraepithelial Lymphocytes Develop in the Intestine and Transform into Lymphomas in Celiac Disease.Interleukin 15 and CD4⁺ T cells cooperate to promote small intestinal enteropathy in response to dietary antigen.Impaired control of effector T cells by regulatory T cells: a clue to loss of oral tolerance and autoimmunity in celiac disease?Enteropathy associated T cell lymphoma in celiac disease: a large retrospective study.Large Granular Lymphocytic Leukemia: A Treatable Form of Refractory Celiac DiseaseEnteropathy-Associated T-Cell Lymphoma Complicating an Autoimmune EnteropathyInhibition of TGF-β Signaling by IL-15: A New Role for IL-15 in the Loss of Immune Homeostasis in Celiac DiseaseEpithelial stress enters the dance in coeliac diseaseLa maladie cœliaque en 2009 : un futur sans régime ?CD28+ intraepithelial lymphocytes with long telomeres are recruited within the inflamed ileal mucosa in Crohn diseaseEpithelial inflammation response induced by Shigella flexneri depends on mucin gene expressionA locus at 7p14.3 predisposes to refractory celiac disease progression from celiac diseaseExpression of nonclassical class I molecules by intestinal epithelial cells
P50
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P50
description
researcher ORCID ID = 0000-0002-8978-8636
@en
wetenschapper
@nl
name
Bertrand Meresse
@ast
Bertrand Meresse
@en
Bertrand Meresse
@es
Bertrand Meresse
@nl
type
label
Bertrand Meresse
@ast
Bertrand Meresse
@en
Bertrand Meresse
@es
Bertrand Meresse
@nl
altLabel
bertrand Meresse
@en
prefLabel
Bertrand Meresse
@ast
Bertrand Meresse
@en
Bertrand Meresse
@es
Bertrand Meresse
@nl
P1053
N-6325-2017
P106
P21
P31
P496
0000-0002-8978-8636