Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer? - Wikidata - awgldk - TriplyDB

Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

http://www.wikidata.org/entity/Q58562408

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

@en

Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

@nl

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TLCR.2018.09.04

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Translational lung cancer research

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Targeting IL22: a potential therapeutic approach for Kras mutant lung cancer?

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Frank D Weinberg

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Nithya Ramnath

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Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine

High fat diet induces formation of spontaneous liposarcoma in mouse adipose tissue with overexpression of interleukin 22

IL-22(+)CD4(+) T cells promote colorectal cancer stemness via STAT3 transcription factor activation and induction of the methyltransferase DOT1L

IL-22 mediates mucosal host defense against Gram-negative bacterial pneumonia.

In vivo consequences of liver-specific interleukin-22 expression in mice: Implications for human liver disease progression.

Interleukin-22 is produced by invariant natural killer T lymphocytes during influenza A virus infection: potential role in protection against lung epithelial damages

Interleukin-22 is elevated in lavage from patients with lung cancer and other pulmonary diseases.

MAP3K3 expression in tumor cells and tumor-infiltrating lymphocytes is correlated with favorable patient survival in lung cancer.

Innate lymphoid cells sustain colon cancer through production of interleukin-22 in a mouse model

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S243-S247

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scholarly article

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10.21037/TLCR.2018.09.04

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Suppl 3

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7

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2018-09-01T00:00:00Z

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30393613

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6193907

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