β-elemene induced anticancer effect in bladder cancer through upregulation of PTEN and suppression of AKT phosphorylation

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β-elemene induced anticancer effect in bladder cancer through upregulation of PTEN and suppression of AKT phosphorylation

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wetenschappelijk artikel

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наукова стаття, опублікована в листопаді 2018

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name

β-elemene induced anticancer e ...... ression of AKT phosphorylation

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β-elemene induced anticancer e ...... ression of AKT phosphorylation

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β-elemene induced anticancer e ...... ression of AKT phosphorylation

@en

β-elemene induced anticancer e ...... ression of AKT phosphorylation

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β-elemene induced anticancer e ...... ression of AKT phosphorylation

@en

β-elemene induced anticancer e ...... ression of AKT phosphorylation

@nl

P1476

β-elemene induced anticancer e ...... ression of AKT phosphorylation

@en

P2093

Bo Cai

http://www.w3.org/2001/XMLSchema#string

Jinxian Pu

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Limin Ma

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Shaojun Nong

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Xin Guo

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You Wu

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P2860

The PI3K/AKT/mTOR pathway is a potential predictor of distinct invasive and migratory capacities in human ovarian cancer cell lines

13,14-bis(cis-3,5-dimethyl-1-piperazinyl)-β-elemene, a novel β-elemene derivative, shows potent antitumor activities via inhibition of mTOR in human breast cancer cells.

β-Elemene treatment of glioblastoma: a single-center retrospective study.

Current clinical regulation of PI3K/PTEN/Akt/mTOR signalling in treatment of human cancer.

β-elemene inhibits tumor-promoting effect of M2 macrophages in lung cancer.

β-Elemene enhances the efficacy of gefitinib on glioblastoma multiforme cells through the inhibition of the EGFR signaling pathway.

Long non-coding RNA LOC572558 inhibits bladder cancer cell proliferation and tumor growth by regulating the AKT-MDM2-p53 signaling axis.

Selective β2-AR Blockage Suppresses Colorectal Cancer Growth Through Regulation of EGFR-Akt/ERK1/2 Signaling, G1-Phase Arrest, and Apoptosis.

β-Elemene inhibits the proliferation of esophageal squamous cell carcinoma by regulating long noncoding RNA-mediated inhibition of hTERT expression.

Altholactone induces reactive oxygen species-mediated apoptosis in bladder cancer T24 cells through mitochondrial dysfunction, MAPK-p38 activation and Akt suppression.

P304

6019-6025

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P31

scholarly article

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10.3892/OL.2018.9401

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P407

English

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P478

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P577

2018-11-01T00:00:00Z

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P698

30333873

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P921

phosphorylation

cancer research

P932

6176406

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