JNK Inactivation Induces Polyploidy and Drug-Resistance in Coronarin D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyploidy and Drug-Resistance in Coronarin D-Treated Osteosarcoma Cells

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im August 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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JNK Inactivation Induces Polyp ...... n D-Treated Osteosarcoma Cells

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Chang-Te Hsu

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Chen-Pu Hsieh

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Chia-Chieh Wu

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Tai-Shan Shen

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Yi-Fu Huang

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P2860

JNK pathway in osteosarcoma: pathogenesis and therapeutics.

JNK-mediated phosphorylation of Cdc25C regulates cell cycle entry and G(2)/M DNA damage checkpoint.

Osteosarcoma Overview.

How Taxol/paclitaxel kills cancer cells.

Activation of c-Jun N-terminal kinase (JNK) during mitosis in retinal progenitor cells.

Advances in cancer therapy with plant based natural products.

The molecular pathogenesis of osteosarcoma: a review

A novel benzimidazole derivative, MBIC inhibits tumor growth and promotes apoptosis via activation of ROS-dependent JNK signaling pathway in hepatocellular carcinoma.

Multinucleated polyploidy drives resistance to Docetaxel chemotherapy in prostate cancer.

Small-molecule inhibitor BMS-777607 induces breast cancer cell polyploidy with increased resistance to cytotoxic chemotherapy agents.

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2121

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scholarly article

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10.3390/MOLECULES23092121

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English

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2018-08-23T00:00:00Z

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30142914

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6225306

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