The expression and clinical significance of GADD45A in breast cancer patients

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The expression and clinical significance of GADD45A in breast cancer patients

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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The expression and clinical significance of GADD45A in breast cancer patients

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The expression and clinical significance of GADD45A in breast cancer patients

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The expression and clinical significance of GADD45A in breast cancer patients

@en

The expression and clinical significance of GADD45A in breast cancer patients

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The expression and clinical significance of GADD45A in breast cancer patients

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The expression and clinical significance of GADD45A in breast cancer patients

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P1476

The expression and clinical significance of GADD45A in breast cancer patients

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P2093

Fei Long

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Fengshang Yan

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Junnan Wang

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Ning Wang

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Yajie Wang

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Yiran Wang

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P2860

Gadd45, a p53-responsive stress protein, modifies DNA accessibility on damaged chromatin

The clinical value of aberrant epigenetic changes of DNA damage repair genes in human cancer

A strong candidate for the breast and ovarian cancer susceptibility gene BRCA1

MiR-148a increases glioma cell migration and invasion by downregulating GADD45A in human gliomas with IDH1 R132H mutations.

Gadd45a functions as a promoter or suppressor of breast cancer dependent on the oncogenic stress

Androgen Receptor (AR), E-Cadherin, and Ki-67 as Emerging Targets and Novel Prognostic Markers in Triple-Negative Breast Cancer (TNBC) Patients

The GADD45A (1506T>C) Polymorphism Is Associated with Ovarian Cancer Susceptibility and Prognosis

Overexpression of DNA damage-induced 45 α gene contributes to esophageal squamous cell cancer by promoter hypomethylation.

P53 and Ki-67 as prognostic markers in triple-negative breast cancer patients.

Gadd45a levels in human breast cancer are hormone receptor dependent

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e5344

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P31

scholarly article

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10.7717/PEERJ.5344

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P407

English

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P577

2018-01-01T00:00:00Z

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30128181

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P932

6098681

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