Exogenous Pancreatic Kallikrein Improves Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetes

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Exogenous Pancreatic Kallikrein Improves Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetes

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im Januar 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована у 2018

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Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

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Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

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Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

@en

Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

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Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

@en

Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

@nl

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Exogenous Pancreatic Kallikrei ...... treptozotocin-Induced Diabetes

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Fangfang Zeng

http://www.w3.org/2001/XMLSchema#string

Ji Hu

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Jie Huang

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Linuo Zhou

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Meng Wang

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Meng Wu

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Min He

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Qin Li

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Shizhe Guo

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Wei Gong

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P2860

Nrf2 is critical in defense against high glucose-induced oxidative damage in cardiomyocytes

Insulin resistance and hyperinsulinaemia in diabetic cardiomyopathy

The kallikrein-kinin system and oxidative stress

Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

SUMO1-dependent modulation of SERCA2a in heart failure.

Role of Oxidative Stress in Thyroid Hormone-Induced Cardiomyocyte Hypertrophy and Associated Cardiac Dysfunction: An Undisclosed Story

Conundrum of pathogenesis of diabetic cardiomyopathy: role of vascular endothelial dysfunction, reactive oxygen species, and mitochondria.

Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.

High glucose inhibits glucose-6-phosphate dehydrogenase, leading to increased oxidative stress and beta-cell apoptosis.

Natriuretic peptides (BNP and NT-proBNP): measurement and relevance in heart failure

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855

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scholarly article

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10.3389/FPHAR.2018.00855

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English

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2018-01-01T00:00:00Z

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30131697

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Farmakologi

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6091235

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