Uric acid-induced endothelial dysfunction is associated with mitochondrial alterations and decreased intracellular ATP concentrations - Wikidata - awgldk - TriplyDB

Uric acid-induced endothelial dysfunction is associated with mitochondrial alterations and decreased intracellular ATP concentrations

Uric acid-induced endothelial dysfunction is associated with mitochondrial alterations and decreased intracellular ATP concentrations

http://www.wikidata.org/entity/Q58865109

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New Pathogenic Concepts and Therapeutic Approaches to Oxidative Stress in Chronic Kidney Disease Uric acid in metabolic syndrome: From an innocent bystander to a central player Mitochondria: a new therapeutic target in chronic kidney disease Uric acid and chronic kidney disease: which is chasing which?Principles of targeting endothelial cell metabolism to treat angiogenesis and endothelial cell dysfunction in disease Role of NADPH Oxidase in Metabolic Disease-Related Renal Injury: An Update Association between perfluoroalkyl acids and kidney function in a cross-sectional study of adolescents Uric Acid Puzzle: Dual Role as Anti-oxidantand Pro-oxidant.High Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ Functions The role of uric acid in kidney fibrosis: experimental evidences for the causal relationship.Protective role of α-lipoic acid in hyperuricemia-induced endothelial dysfunction.Hyperuricemia as a Predictive Marker for Progression of Nephrosclerosis: Clinical Assessment of Prognostic Factors in Biopsy-Proven Arterial/Arteriolar Nephrosclerosis Increased Serum Uric Acid Level Is a Risk Factor for Left Ventricular Hypertrophy but Not Independent of eGFR in Patients with Type 2 Diabetic Kidney Disease Sugar, uric acid, and the etiology of diabetes and obesity.Comorbidities in patients with crystal diseases and hyperuricemia.Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.Uric acid in the pathogenesis of metabolic, renal, and cardiovascular diseases: A review.Uric acid promotes apoptosis in human proximal tubule cells by oxidative stress and the activation of NADPH oxidase NOX 4.Association of serum uric acid and incident nonspine fractures in elderly men: the Osteoporotic Fractures in Men (MrOS) study.Renal oxidative stress induced by long-term hyperuricemia alters mitochondrial function and maintains systemic hypertension.Uric Acid as a Marker of Mortality and Morbidity in Fabry Disease.Perspective: A Historical and Scientific Perspective of Sugar and Its Relation with Obesity and Diabetes.Fructose and uric acid in diabetic nephropathy Uric acid and transforming growth factor in fructose-induced production of reactive oxygen species in skeletal muscle The mechanisms underlying fructose-induced hypertension: a review Protective effects of aspirin and vitamin C against corn syrup consumption-induced cardiac damage through sirtuin-1 and HIF-1α pathway.The role of uric acid in the pathogenesis of human cardiovascular disease.Hyperuricemia-induced NLRP3 activation of macrophages contributes to the progression of diabetic nephropathy.Time to target uric acid to retard CKD progression.Uric acid enhances PKC-dependent eNOS phosphorylation and mediates cellular ER stress: A mechanism for uric acid-induced endothelial dysfunction.Activation of Renal (Pro)Renin Receptor Contributes to High Fructose-Induced Salt Sensitivity.Diabetic nephropathy: Time to withhold development and progression - A review.Relevance of uric acid and asymmetric dimethylarginine for modeling cardiovascular risk prediction in chronic kidney disease patients.Podocyte Injury and Albuminuria in Experimental Hyperuricemic Model Rats.Uric Acid Induces Endothelial Dysfunction by Activating the HMGB1/RAGE Signaling Pathway Is gout a risk equivalent to diabetes for stroke and myocardial infarction? A retrospective claims database study.Direct and Indirect Effects of Serum Uric Acid on Blood Sugar Levels in Patients with Prediabetes: A Mediation Analysis.Effect of Febuxostat on Ambulatory Blood Pressure in Subjects With Hyperuricemia and Hypertension: A Phase 2 Randomized Placebo-Controlled Study.Uric Acid and the Risks of Kidney Failure and Death in Individuals With CKD.Novel treatment strategies for chronic kidney disease: insights from the animal kingdom.

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Uric acid-induced endothelial dysfunction is associated with mitochondrial alterations and decreased intracellular ATP concentrations

http://www.wikidata.org/entity/Q58865109

description

im Dezember 2012 veröffentlichter wissenschaftlicher Artikel

@de

scientific article published on 07 December 2012

@en

wetenschappelijk artikel

@nl

наукова стаття, опублікована у 2012

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name

Uric acid-induced endothelial ...... tracellular ATP concentrations

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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type

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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The Nephron Journals

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Uric acid-induced endothelial ...... tracellular ATP concentrations

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Duk-Hee Kang

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Fernando García-Arroyo

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Magdalena Cristóbal-García

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Miguel A Lanaspa

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Min A Yu

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Richard J Johnson

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Takahiko Nakagawa

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Virgilia Soto

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P2860

Sugar-sweetened beverages and risk of metabolic syndrome and type 2 diabetes: a meta-analysis

Sugar-Sweetened Beverages, Obesity, Type 2 Diabetes Mellitus, and Cardiovascular Disease Risk

Intake of sugar-sweetened beverages and weight gain: a systematic review

The Krebs cycle and mitochondrial mass are early victims of endothelial dysfunction: proteomic approach.

Sucrose-sweetened beverages increase fat storage in the liver, muscle, and visceral fat depot: a 6-mo randomized intervention study.

Enoyl-CoA hydratase. reaction, mechanism, and inhibition.

A causal role for uric acid in fructose-induced metabolic syndrome.

Role of angiotensin II and endogenous vasodilators in the control of glomerular hemodynamics.

Short-term alterations in carbohydrate energy intake in humans. Striking effects on hepatic glucose production, de novo lipogenesis, lipolysis, and whole-body fuel selection.

Ketohexokinase-dependent metabolism of fructose induces proinflammatory mediators in proximal tubular cells.

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e71-8

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scholarly article

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10.1159/000345509

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3-4

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121

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David Cruz-Robles

Laura Gabriela Sánchez-Lozada

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2012-12-07T00:00:00Z

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