about
AhR-Mediated, Non-Genomic Modulation of IDO1 FunctionIndoleamine 2,3-dioxygenase is a signaling protein in long-term tolerance by dendritic cellsAryl hydrocarbon receptor control of a disease tolerance defence pathwayForced IDO1 expression in dendritic cells restores immunoregulatory signalling in autoimmune diabetesStem cells from human amniotic fluid exert immunoregulatory function via secreted indoleamine 2,3-dioxygenase1.IDO1 and TGF-β Mediate Protective Effects of IFN-α in Antigen-Induced Arthritis.LPS-conditioned dendritic cells confer endotoxin tolerance contingent on tryptophan catabolism.Cutting edge: Autocrine TGF-beta sustains default tolerogenesis by IDO-competent dendritic cells.High doses of CpG oligodeoxynucleotides stimulate a tolerogenic TLR9-TRIF pathway.Immunosuppression via tryptophan catabolism: the role of kynurenine pathway enzymesImmunohistochemistry predicts nucleophosmin (NPM) mutations in acute myeloid leukemiaA western blot assay for detecting mutant nucleophosmin (NPM1) proteins in acute myeloid leukaemia
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-0417-1638
@en
name
Maria T Pallotta
@ast
Maria T Pallotta
@en
Maria T Pallotta
@es
Maria T Pallotta
@nl
type
label
Maria T Pallotta
@ast
Maria T Pallotta
@en
Maria T Pallotta
@es
Maria T Pallotta
@nl
prefLabel
Maria T Pallotta
@ast
Maria T Pallotta
@en
Maria T Pallotta
@es
Maria T Pallotta
@nl
P106
P31
P496
0000-0002-0417-1638