about
Repair on the go: E. coli maintains a high proliferation rate while repairing a chronic DNA double-strand breakATR-ATRIP kinase complex triggers activation of the Fanconi anemia DNA repair pathway.DNA tandem repeat instability in the Escherichia coli chromosome is stimulated by mismatch repair at an adjacent CAG·CTG trinucleotide repeatE. coli SbcCD and RecA control chromosomal rearrangement induced by an interrupted palindrome.Co-mutation of histone H2AX S139A with Y142A rescues Y142A-induced ionising radiation sensitivity.ATR activates the S-M checkpoint during unperturbed growth to ensure sufficient replication prior to mitotic onset.Non-random segregation of sister chromosomes in Escherichia coli.Phenotypic and functional heterogeneity of human intermediate monocytes based on HLA-DR expression.SbcCD causes a double-strand break at a DNA palindrome in the Escherichia coli chromosome.Analysis of novel missense ATR mutations reveals new splicing defects underlying Seckel syndromeLive imaging of marked chromosome regions reveals their dynamic resolution and compaction in mitosisContractile acto-myosin network on nuclear envelope remnants positions human chromosomes for mitosisZooming in on chromosome dynamics
P50
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P50
description
researcher ORCID ID = 0000-0003-4115-9686
@en
wetenschapper
@nl
name
John K Eykelenboom
@ast
John K Eykelenboom
@en
John K Eykelenboom
@es
John K Eykelenboom
@nl
type
label
John K Eykelenboom
@ast
John K Eykelenboom
@en
John K Eykelenboom
@es
John K Eykelenboom
@nl
prefLabel
John K Eykelenboom
@ast
John K Eykelenboom
@en
John K Eykelenboom
@es
John K Eykelenboom
@nl
P106
P31
P496
0000-0003-4115-9686