DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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Genomic variation at the tips of the adaptive radiation of Darwin's finches.

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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наукова стаття, опублікована у 2013

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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DLK1 Protein Expression during Mouse Development Provides New Insights into Its Function

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D. C. Aronson

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F. A. Falix

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I. C. Gaemers

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M. R. S. Tjon-A-Loi

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W. H. Lamers

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P2860

Expression patterns of Jagged, Delta1, Notch1, Notch2, and Notch3 genes identify ligand-receptor pairs that may function in neural development

High delta-like 1 expression in a subset of neuroblastoma cell lines corresponds to a differentiated chromaffin cell type

Pref-1, a protein containing EGF-like repeats, inhibits adipocyte differentiation

Studies on the isolation, structural analysis and tissue localization of fetal antigen 1 and its relation to a human adrenal-specific cDNA, pG2

Notch signalling controls pancreatic cell differentiation

Control of endodermal endocrine development by Hes-1

Sustained Notch signaling in progenitors is required for sequential emergence of distinct cell lineages during organogenesis

The EGF-like protein dlk1 inhibits notch signaling and potentiates adipogenesis of mesenchymal cells

Activated Notch1 prevents differentiation of pancreatic acinar cells and attenuate endocrine development

The canonical Notch signaling pathway: unfolding the activation mechanism.

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1-10

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scholarly article

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10.1155/2013/628962

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2013

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2013-01-01T00:00:00Z

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