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Plasmin cleavage of von Willebrand factor as an emergency bypass for ADAMTS13 deficiency in thrombotic microangiopathy.Quantification of platelet-surface interactions in real-time using intracellular calcium signaling.Variation at the paraoxonase gene locus contributes to carotid arterial wall thickness in subjects with familial hypercholesterolemia.Polyphosphate nanoparticles on the platelet surface trigger contact system activation.Preeclampsia and coronary plaque erosion: Manifestations of endothelial dysfunction resulting in cardiovascular events in women.Live-cell Imaging of Platelet Degranulation and Secretion Under Flow.Interaction of Extracellular Vesicles with Endothelial Cells Under Physiological Flow Conditions.Platelet function is disturbed by the angiogenesis inhibitors sunitinib and sorafenib, but unaffected by bevacizumab.Quantitative proteomics analysis reveals similar release profiles following specific PAR-1 or PAR-4 stimulation of plateletsGenetic and environmental determinants of the PON-1 phenotypeRisk of acute ischemic heart disease in postmenopausal women depends on von Willebrand factor and fibrinogen concentrations, and blood group genotypeResponse to Letter Regarding Article, “Plasmin Cleavage of von Willebrand Factor as an Emergency Bypass for ADAMTS13 Deficiency in Thrombotic Microangiopathy”Plasma concentration of von Willebrand factor predicts mortality in patients on chronic renal replacement therapyHigh-throughput genotyping with infrared fluorescence allele specific hybridization (iFLASH): a simple, reliable and low-cost alternativeHeparin Forms Polymers with Cell-free DNA Which Elongate Under Shear in Flowing Blood
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P50
description
researcher ORCID ID = 0000-0002-0653-5538
@en
wetenschapper
@nl
name
Arjan D Barendrecht
@ast
Arjan D Barendrecht
@en
Arjan D Barendrecht
@nl
type
label
Arjan D Barendrecht
@ast
Arjan D Barendrecht
@en
Arjan D Barendrecht
@nl
prefLabel
Arjan D Barendrecht
@ast
Arjan D Barendrecht
@en
Arjan D Barendrecht
@nl
P31
P496
0000-0002-0653-5538