about
Single dose of acetylsalicylic acid in patients with Type 2 diabetes mellitus and/or chronic renal failure ameliorates anaemia by decreasing the rate of neocytolysisAlpha-1 antitrypsin binds preprohepcidin intracellularly and prohepcidin in the serumGeographic differences in genetic susceptibility to IgA nephropathy: GWAS replication study and geospatial risk analysis.Discovery of new risk loci for IgA nephropathy implicates genes involved in immunity against intestinal pathogens.A history of diabetes mellitus or how a disease of the kidneys evolved into a kidney disease.Metabolic syndrome and other cardiovascular risk factors associated with the progression of IgA nephropathy.Influence of body mass index on the association of weight changes with mortality in hemodialysis patientsProhepcidin binds to the HAMP promoter and autoregulates its own expression.Microalbuminuria, indicated by total versus immunoreactive urinary albumins, in acute ischemic stroke patients.Different effect of IgA nephropathy and polycystic kidney disease on arterial stiffness.Role of iron in the interaction of red blood cells with methylglyoxal. Modification of L-arginine by methylglyoxal is catalyzed by iron redox cycling.COSMOS: the dialysis scenario of CKD-MBD in Europe.Cigarette smoke and its formaldehyde component inhibit bradykinin-induced calcium increase in pig aortic endothelial cells.Serum levels of L-arginine analogues and glutathione peroxidase and catalase gene variants in Type 2 diabetes mellitus patients.[The effect of low-protein diet supplemented with ketoacids in patients with chronic renal failure].Serum total LDH activity and LDH-2 isozyme in nephrotic syndrome.Richard Bright in Hungary: a reevaluation.Polymorphisms of the IL23R gene are associated with psoriasis but not with immunoglobulin A nephropathy in a Hungarian population.A second field metachronous Merkel cell carcinoma of the lip and the palatine tonsil confirmed by microarray-based comparative genomic hybridisation.Use of phosphate-binding agents is associated with a lower risk of mortalityIgA glomerulonephritis. Mesangial IgA deposition without systemic signs (Berger's disease)Plasma factors influencing PGI2-like activity in patients with IgA nephropathy and Henoch-Schönlein purpuraThe effects of plasma from IgA nephropathy patients on vascular prostacyclin and platelet cyclic AMP productionAntibodies to viruses of the herpes group in glomerulonephritis of membranous, membranoproliferative and IgA typesThe role of the tubulointerstitial changes in the prognosis of IgA glomerulonephritisAre insulin resistance and atherosclerosis the consequences of oxidative stress?Do intestinal hyperpermeability and the related food antigens play a role in the progression of IgA nephropathy? I. Study of intestinal permeabilityOxidative stress and antioxidant defense mechanism in glomerular diseasesPossible pathogenic role of IgE in Henoch-Schönlein purpuraEffect of CP-96,345 a non-peptide substance P antagonist, capsaicin, resiniferatoxin and ruthenium red on nociceptionHypercholesterolemia promotes endothelial dysfunction in vitamin E- and selenium-deficient ratsMembrane attack complex and membrane cofactor protein are related to tubulointerstitial inflammation in various human glomerulopathiesAn unusual multiplex cause of severe gastrointestinal bleeding in a haemodialysed patientCircadian blood pressure changes and cardiac abnormalities in IgA nephropathyThe kinetics of initial gastrointestinal absorption of an aluminium-containing antacid (Tisacid) in patients with various stages of chronic renal insufficiencySpecific von Hippel-Lindau protein expression of clear cell renal cell carcinoma with "immunogenic" featuresPossible role of free radicals generated by pseudohypoxia in the regulation of hepatic glucose output. An in vitro model using rat liver microsomal glucose 6-phosphataseAssociation of a uteroglobin polymorphism with rate of progression in patients with IgA nephropathyAmbulatory blood pressure monitoring and progression in patients with IgA nephropathyInduction of endothelial cell injury by cigarette smoke
P50
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P50
description
researcher ORCID ID = 0000-0003-1702-5071
@en
wetenschapper
@nl
name
Judit Nagy
@ast
Judit Nagy
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Judit Nagy
@es
Judit Nagy
@nl
type
label
Judit Nagy
@ast
Judit Nagy
@en
Judit Nagy
@es
Judit Nagy
@nl
prefLabel
Judit Nagy
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Judit Nagy
@en
Judit Nagy
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Judit Nagy
@nl
P1153
7202084133
P31
P496
0000-0003-1702-5071