about
Skeletal muscle PGC-1α controls whole-body lactate homeostasis through estrogen-related receptor α-dependent activation of LDH B and repression of LDH ASkeletal muscle homeostasis and plasticity in youth and ageing: impact of nutrition and exerciseUnderstanding the acetylome: translating targeted proteomics into meaningful physiologyThe coactivator PGC-1α regulates skeletal muscle oxidative metabolism independently of the nuclear receptor PPARβ/δ in sedentary mice fed a regular chow diet.The corepressor NCoR1 antagonizes PGC-1α and estrogen-related receptor α in the regulation of skeletal muscle function and oxidative metabolism.The transcriptional coactivator PGC-1α is dispensable for chronic overload-induced skeletal muscle hypertrophy and metabolic remodeling.Pathophysiological relevance of the cardiac β2-adrenergic receptor and its potential as a therapeutic target to improve cardiac function.Nutritional strategies to support concurrent training.Regulation of skeletal muscle mitochondrial function by nuclear receptors: implications for health and disease.Overload-mediated skeletal muscle hypertrophy is not impaired by loss of myofiber STAT3.Pyruvate suppresses PGC1alpha expression and substrate utilization despite increased respiratory chain content in C2C12 myotubesRapamycin does not prevent increases in myofibrillar or mitochondrial protein synthesis following endurance exercise.PDE2 activity differs in right and left rat ventricular myocardium and differentially regulates β2 adrenoceptor-mediated effects.Exercise and high-fat feeding remodel transcript-metabolite interactive networks in mouse skeletal muscle.Over-expression of a retinol dehydrogenase (SRP35/DHRS7C) in skeletal muscle activates mTORC2, enhances glucose metabolism and muscle performance.Regulation of contractility and metabolic signaling by the β2-adrenergic receptor in rat ventricular muscle.HIF1A P582S gene association with endurance training responses in young womenVariability in the magnitude of response of metabolic enzymes reveals patterns of co-ordinated expression following endurance training in womenSingle inhibition of either PDE3 or PDE4 unmasks β2-adrenoceptor-mediated inotropic and lusitropic effects in the left but not right ventricular myocardium of rat
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description
researcher ORCID ID = 0000-0003-3141-8859
@en
wetenschapper
@nl
name
Joaquín Pérez-Schindler
@ast
Joaquín Pérez-Schindler
@en
Joaquín Pérez-Schindler
@nl
type
label
Joaquín Pérez-Schindler
@ast
Joaquín Pérez-Schindler
@en
Joaquín Pérez-Schindler
@nl
prefLabel
Joaquín Pérez-Schindler
@ast
Joaquín Pérez-Schindler
@en
Joaquín Pérez-Schindler
@nl
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0000-0003-3141-8859