about
Bidirectional TSH and IGF-1 receptor cross talk mediates stimulation of hyaluronan secretion by Graves' disease immunoglobinsFuture Prospects for the Treatment of Graves' Hyperthyroidism and Eye Disease.PTH and Vitamin D Repress DMP1 in Cementoblasts.A modified ELISA accurately measures secretion of high molecular weight hyaluronan (HA) by Graves' disease orbital cells.Inhibiting thyrotropin/insulin-like growth factor 1 receptor crosstalk to treat Graves' ophthalmopathy: studies in orbital fibroblasts in vitro.Forced unfolding of proteins within cells.Heart-specific stiffening in early embryos parallels matrix and myosin expression to optimize beating.TSH/IGF-1 Receptor Cross-Talk Rapidly Activates Extracellular Signal-Regulated Kinases in Multiple Cell Types.TSHR/IGF-1R Cross-Talk, Not IGF-1R Stimulating Antibodies, Mediates Graves' Ophthalmopathy Pathogenesis.Evidence That Graves' Ophthalmopathy Immunoglobulins Do Not Directly Activate IGF-1 ReceptorsThyroid Stimulating Hormone (TSH)/Insulin-like Growth Factor 1 (IGF1) Receptor Cross-talk in Human CellsLetter to the Editor: "Elevated Serum Tetrac in Graves Disease: Potential Pathogenic Role in Thyroid-Associated Ophthalmopathy"Arrestin-β-1 Physically Scaffolds TSH and IGF1 Receptors to Enable Crosstalk
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-1856-415X
@en
name
Christine Krieger
@ast
Christine Krieger
@en
Christine Krieger
@es
Christine Krieger
@nl
type
label
Christine Krieger
@ast
Christine Krieger
@en
Christine Krieger
@es
Christine Krieger
@nl
prefLabel
Christine Krieger
@ast
Christine Krieger
@en
Christine Krieger
@es
Christine Krieger
@nl
P106
P21
P31
P496
0000-0002-1856-415X