about
Site-specific chemokine expression regulates central nervous system inflammation and determines clinical phenotype in autoimmune encephalomyelitisCritical role for Ipaf in Pseudomonas aeruginosa-induced caspase-1 activation.Notch signaling regulates T cell accumulation and function in the central nervous system during experimental autoimmune encephalomyelitis.TLR9-induced interferon β is associated with protection from gammaherpesvirus-induced exacerbation of lung fibrosis.GM-CSF Promotes Chronic Disability in Experimental Autoimmune Encephalomyelitis by Altering the Composition of Central Nervous System-Infiltrating Cells, but Is Dispensable for Disease Induction.
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description
researcher ORCID ID = 0000-0003-3065-9300
@en
wetenschapper
@nl
name
Joshua Stoolman
@ast
Joshua Stoolman
@en
Joshua Stoolman
@es
Joshua Stoolman
@nl
type
label
Joshua Stoolman
@ast
Joshua Stoolman
@en
Joshua Stoolman
@es
Joshua Stoolman
@nl
prefLabel
Joshua Stoolman
@ast
Joshua Stoolman
@en
Joshua Stoolman
@es
Joshua Stoolman
@nl
P106
P31
P496
0000-0003-3065-9300